The failing heart is a major source of circulating FGF23 via oncostatin M receptor activation

J Heart Lung Transplant. 2015 Sep;34(9):1211-4. doi: 10.1016/j.healun.2015.06.007. Epub 2015 Jun 17.

Authors

Manfred Richter¹, Hans-Jürgen Lautze², Thomas Walther¹, Thomas Braun³, Sawa Kostin³, Thomas Kubin³

Affiliations

¹ Departments of Cardiac Surgery.
² Anesthesiology, Kerckhoff-Clinic, Benekestrasse, Bad Nauheim, Germany.
³ Department of Cardiac Development and Remodeling, Max-Planck-Institute for Heart and Lung Research, Bad Nauheim, Germany.

PMID: 26267742
DOI: 10.1016/j.healun.2015.06.007

No abstract available

Publication types

Letter
Research Support, Non-U.S. Gov't

MeSH terms

Animals
Cells, Cultured
Fibroblast Growth Factor-23
Fibroblast Growth Factors / blood
Fibroblast Growth Factors / metabolism*
Heart Failure / physiopathology*
Humans
Mice
Receptors, Oncostatin M / physiology*

Substances

FGF23 protein, human
Fgf23 protein, mouse
Receptors, Oncostatin M
Fibroblast Growth Factors
Fibroblast Growth Factor-23