Editorial Comment: Does gut-derived inflammation enhance pain signaling following chemotherapy in a Toll-like receptor 4-dependent manner?

Curr Opin Support Palliat Care. 2015 Jun;9(2):155-6. doi: 10.1097/SPC.0000000000000139.

Authors

Hannah R Wardill¹, Ysabella Z A Van Sebille, Joanne M Bowen, Rachel J Gibson

Affiliation

¹ aDiscipline of Anatomy and Pathology bDiscipline of Physiology, School of Medical Sciences, University of Adelaide, South Australia, Australia *Hannah R. Wardill and Ysabella Z.A. Van Sebille contributed equally to the writing of this article.

PMID: 25872125
DOI: 10.1097/SPC.0000000000000139

No abstract available

Publication types

Editorial

MeSH terms

Antineoplastic Agents / adverse effects*
Antineoplastic Agents / immunology
Antineoplastic Agents / therapeutic use
Humans
Inflammation / complications
Inflammation / physiopathology*
Intestines / immunology*
Intestines / physiopathology
Neoplasms / complications
Neoplasms / drug therapy
Neuroglia / immunology*
Neuroglia / physiology
Pain / chemically induced*
Pain / immunology
Pain / physiopathology
Signal Transduction / immunology
Toll-Like Receptor 4 / immunology*
Toll-Like Receptor 4 / physiology

Substances

Antineoplastic Agents
TLR4 protein, human
Toll-Like Receptor 4