Exchanging cardiac phenotype: Is AKT-mediated NHE1 inhibition a permissive switch in physiological hypertrophy?

J Mol Cell Cardiol. 2014 Dec:77:175-7. doi: 10.1016/j.yjmcc.2014.10.007. Epub 2014 Oct 23.
No abstract available

Publication types

  • Editorial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cation Transport Proteins / metabolism*
  • Heart / physiology*
  • Humans
  • Hypertrophy / enzymology
  • Phenotype
  • Proto-Oncogene Proteins c-akt / physiology*
  • Sodium-Hydrogen Exchanger 1
  • Sodium-Hydrogen Exchangers / metabolism*

Substances

  • Cation Transport Proteins
  • SLC9A1 protein, human
  • Sodium-Hydrogen Exchanger 1
  • Sodium-Hydrogen Exchangers
  • Proto-Oncogene Proteins c-akt