Socs3 induction by PPARγ restrains cancer-promoting inflammation

Cell Cycle. 2013 Jul 15;12(14):2157-8. doi: 10.4161/cc.25370.
No abstract available

Publication types

  • Editorial

MeSH terms

  • Animals
  • Cell Differentiation / drug effects
  • Cell Transformation, Neoplastic / genetics*
  • Cell Transformation, Neoplastic / metabolism
  • Cell Transformation, Neoplastic / pathology
  • Cells, Cultured
  • Docosahexaenoic Acids / pharmacology
  • Gene Expression Regulation
  • Humans
  • Inflammation / genetics
  • Inflammation / metabolism
  • Inflammation / pathology
  • Interleukin-17 / biosynthesis*
  • Interleukin-17 / metabolism
  • Mice
  • PPAR gamma / genetics*
  • PPAR gamma / metabolism
  • Phosphorylation
  • Promoter Regions, Genetic
  • Protein Binding
  • STAT3 Transcription Factor / genetics
  • STAT3 Transcription Factor / metabolism
  • Signal Transduction
  • Suppressor of Cytokine Signaling 3 Protein
  • Suppressor of Cytokine Signaling Proteins / genetics*
  • Suppressor of Cytokine Signaling Proteins / metabolism
  • Th17 Cells / cytology
  • Th17 Cells / drug effects
  • Th17 Cells / metabolism*

Substances

  • Interleukin-17
  • PPAR gamma
  • SOCS3 protein, human
  • STAT3 Transcription Factor
  • STAT3 protein, human
  • Suppressor of Cytokine Signaling 3 Protein
  • Suppressor of Cytokine Signaling Proteins
  • Docosahexaenoic Acids