'I feel bad', 'We feel good'?--emotions as a driver for personal and organizational identity and organizational identification as a resource for serving unfriendly customers

Stress Health. 2012 Apr;28(2):123-36. doi: 10.1002/smi.1412. Epub 2011 Jul 20.

Abstract

The social identity approach is used to demonstrate how personal and organizational identity is affected by emotions at work and that organizational identification can function as a valuable resource in coping with stressors. We analysed data from an experiment with 96 call centre agents to investigate relationships between positive and negative emotions, identification and strain. Positive and negative emotions were induced by simulated customers who either behaved in a friendly or a rude way. Organizational identification was assessed with a questionnaire, and personal identity salience was measured using video data by counting how often agents said 'I' during conversations. Strain was measured through self-reports of emotional dissonance and by assessing immunoglobulin A (IgA) concentration in participants' saliva. Results showed that organizational identification was higher in conditions with positive emotions and was reduced by the induction of negative emotions. Moreover, organizational identification functioned as a buffer against stress: emotional dissonance generally was lower for participants with high organizational identification; IgA levels were negatively associated with organizational identification when agents communicated with unfriendly customers. Conversely, personal identity salience was induced by negative emotions and did not make a positive contribution to the coping process.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptation, Psychological
  • Adult
  • Emotions*
  • Female
  • Humans
  • Immunoglobulin A / analysis
  • Interpersonal Relations
  • Male
  • Occupational Health
  • Organizational Culture
  • Saliva / chemistry
  • Self Concept*
  • Social Identification*
  • Stress, Psychological / etiology*
  • Telephone

Substances

  • Immunoglobulin A