S-propargyl-cysteine, a novel hydrogen sulfide-modulated agent, attenuated tumor necrosis factor-α-induced inflammatory signaling and dysfunction in endothelial cells

Int J Cardiol. 2012 Mar 8;155(2):327-32. doi: 10.1016/j.ijcard.2011.12.059. Epub 2012 Jan 10.
No abstract available

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Atherosclerosis / drug therapy*
  • Atherosclerosis / immunology
  • Cysteine / analogs & derivatives*
  • Cysteine / pharmacology
  • Endothelial Cells / drug effects
  • Endothelial Cells / immunology
  • Garlic
  • Human Umbilical Vein Endothelial Cells
  • Humans
  • Hydrogen Sulfide / pharmacology
  • Signal Transduction / drug effects*
  • Signal Transduction / immunology*
  • Tumor Necrosis Factor-alpha / immunology*
  • Tumor Necrosis Factor-alpha / pharmacology
  • U937 Cells
  • Vasculitis / drug therapy*
  • Vasculitis / immunology

Substances

  • S-propargylcysteine
  • Tumor Necrosis Factor-alpha
  • Cysteine
  • Hydrogen Sulfide