Mitochondrial dysfunction and neurodegenerative diseases

World Neurosurg. 2010 Jul;74(1):10-2. doi: 10.1016/j.wneu.2010.08.015.
No abstract available

Publication types

  • News

MeSH terms

  • Animals
  • Animals, Genetically Modified
  • Apoptosis / genetics
  • Apoptosis / physiology
  • Axonal Transport / genetics
  • Axonal Transport / physiology
  • Cell Line
  • Disease Models, Animal
  • Endoplasmic Reticulum / physiology
  • Energy Metabolism / genetics
  • Energy Metabolism / physiology
  • Free Radicals
  • Humans
  • Huntingtin Protein
  • Mitochondrial Diseases / genetics
  • Mitochondrial Diseases / physiopathology*
  • Mutation
  • Nerve Tissue Proteins / genetics
  • Nerve Tissue Proteins / physiology
  • Neurodegenerative Diseases / genetics
  • Neurodegenerative Diseases / physiopathology*
  • Neurons / physiology
  • Nuclear Proteins / genetics
  • Nuclear Proteins / physiology
  • Oxidative Stress / genetics
  • Oxidative Stress / physiology
  • Protein Unfolding
  • tau Proteins / genetics
  • tau Proteins / physiology

Substances

  • Free Radicals
  • HTT protein, human
  • Huntingtin Protein
  • Nerve Tissue Proteins
  • Nuclear Proteins
  • tau Proteins