DNA damage induced by shikonin in the presence of Cu(II) ions: potential mechanism of its activity to apoptotic cell death

J Asian Nat Prod Res. 2011 Jan;13(1):12-9. doi: 10.1080/10286020.2010.537262.

Abstract

Shikonin (β-alkannin), a naphthazarin derivative, has shown a variety of abilities such as anti-inflammatory, antitumoral, cytotoxic, and antimicrobial activities. In the presence of Cu(II), shikonin caused breakage of supercoiled plasmid pBR322 DNA. Other metal ions tested [Mg(II), Ca(II), and Ni(II)] were ineffective and only Fe(II) has the same ability in the DNA breakage reaction. The involvement of active oxygen in the reaction was established by the inhibition of DNA breakage by superoxide dismutase, catalase, thiourea, sodium azide, potassium iodide, and sodium benzoate. Cu(I) was shown to be an essential intermediate using the Cu(I)-specific sequestering reagent neocuproine. Shikonin induced HeLa cell apoptosis involved in the mechanism of increasing intracellular reactive oxygen species (ROS). It was suggested that shikonin generated ROS as a pro-oxidant in the presence of Cu(II), and ROS resulted in DNA damage and apoptotic cell death in cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / drug effects*
  • Copper / pharmacology*
  • DNA Damage*
  • Drugs, Chinese Herbal / pharmacology
  • Drugs, Chinese Herbal / toxicity*
  • HeLa Cells
  • Humans
  • Molecular Structure
  • Naphthoquinones / pharmacology
  • Naphthoquinones / toxicity*
  • Oxidation-Reduction
  • Plasmids / genetics
  • Reactive Oxygen Species / toxicity

Substances

  • Drugs, Chinese Herbal
  • Naphthoquinones
  • Reactive Oxygen Species
  • shikonin
  • Copper