Histamine modulates the responsiveness of keratinocytes to IL-17 and TNF-α through the H1-receptor

J Dermatol Sci. 2011 Jan;61(1):79-81. doi: 10.1016/j.jdermsci.2010.11.005. Epub 2010 Nov 13.
No abstract available

Publication types

  • Letter

MeSH terms

  • Butyrophenones / pharmacology
  • Cells, Cultured
  • Granulocyte-Macrophage Colony-Stimulating Factor / metabolism
  • Histamine / physiology*
  • Histamine H1 Antagonists / pharmacology
  • Humans
  • Inflammation / physiopathology
  • Inflammation Mediators / metabolism*
  • Inflammation Mediators / physiology
  • Interleukin-17 / physiology*
  • Interleukin-8 / metabolism
  • Keratinocytes / immunology
  • Keratinocytes / metabolism*
  • Keratinocytes / physiology
  • Piperidines / pharmacology
  • Receptors, Histamine H1 / drug effects
  • Receptors, Histamine H1 / physiology*
  • Tumor Necrosis Factor-alpha / physiology*

Substances

  • Butyrophenones
  • Histamine H1 Antagonists
  • Inflammation Mediators
  • Interleukin-17
  • Interleukin-8
  • Piperidines
  • Receptors, Histamine H1
  • Tumor Necrosis Factor-alpha
  • carebastine
  • Histamine
  • Granulocyte-Macrophage Colony-Stimulating Factor