Cigarette smoking induces vascular proliferative disease through the activation of Egr-1

Cardiovasc Res. 2010 Nov 1;88(2):207-8. doi: 10.1093/cvr/cvq295. Epub 2010 Sep 10.
No abstract available

Publication types

  • Comment
  • Editorial

MeSH terms

  • Administration, Oral
  • Animals
  • Atherosclerosis / etiology*
  • Atherosclerosis / metabolism
  • Atherosclerosis / pathology
  • Atherosclerosis / prevention & control
  • Calcium Signaling / drug effects
  • Cell Proliferation / drug effects*
  • Early Growth Response Protein 1 / genetics
  • Early Growth Response Protein 1 / metabolism*
  • Enzyme Activation
  • Humans
  • Iliac Artery / drug effects
  • Iliac Artery / metabolism
  • Iliac Artery / pathology
  • Mitogen-Activated Protein Kinase 1 / antagonists & inhibitors
  • Mitogen-Activated Protein Kinase 1 / metabolism
  • Mitogen-Activated Protein Kinase 3 / antagonists & inhibitors
  • Mitogen-Activated Protein Kinase 3 / metabolism
  • Muscle, Smooth, Vascular / drug effects*
  • Muscle, Smooth, Vascular / injuries
  • Muscle, Smooth, Vascular / metabolism
  • Muscle, Smooth, Vascular / pathology
  • Nicotine / administration & dosage
  • Nicotine / adverse effects
  • Nicotinic Agonists / administration & dosage
  • Nicotinic Agonists / adverse effects
  • Phosphorylation
  • Rats
  • Receptors, Nicotinic / drug effects
  • Receptors, Nicotinic / metabolism
  • Smoking / adverse effects*
  • Tunica Intima / drug effects*
  • Tunica Intima / injuries
  • Tunica Intima / metabolism
  • Tunica Intima / pathology

Substances

  • Early Growth Response Protein 1
  • Egr1 protein, rat
  • Nicotinic Agonists
  • Receptors, Nicotinic
  • Nicotine
  • Mitogen-Activated Protein Kinase 1
  • Mitogen-Activated Protein Kinase 3