Loss of inhibitory insulin receptor substrate-1 phosphorylation: An early event in endometrial hyperplasia and progression to carcinoma

Cell Cycle. 2010 Jul 15;9(14):2698-9. doi: 10.4161/cc.9.14.12618. Epub 2010 Jul 9.
No abstract available

Publication types

  • Editorial
  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Carcinoma / etiology*
  • Carcinoma / metabolism
  • Disease Models, Animal
  • Endometrial Hyperplasia / complications*
  • Endometrial Hyperplasia / metabolism
  • Endometrial Neoplasms / etiology*
  • Endometrial Neoplasms / metabolism
  • Female
  • Humans
  • Insulin Receptor Substrate Proteins / metabolism*
  • Mechanistic Target of Rapamycin Complex 1
  • Multiprotein Complexes
  • Obesity / complications
  • Obesity / metabolism
  • Phosphorylation
  • Proteins / metabolism
  • Rats
  • Signal Transduction
  • TOR Serine-Threonine Kinases
  • Tuberous Sclerosis Complex 2 Protein
  • Tumor Suppressor Proteins / metabolism

Substances

  • Insulin Receptor Substrate Proteins
  • Multiprotein Complexes
  • Proteins
  • Tuberous Sclerosis Complex 2 Protein
  • Tumor Suppressor Proteins
  • Mechanistic Target of Rapamycin Complex 1
  • TOR Serine-Threonine Kinases