Repression of FasL expression by dipfluzine involves a mechanism of inhibition of NFATc transactivation

Yao Xue Xue Bao. 2008 Dec;43(12):1258-63.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Binding Sites
  • Calcium Channel Blockers / pharmacology
  • Cinnarizine / analogs & derivatives*
  • Cinnarizine / pharmacology
  • Fas Ligand Protein / genetics
  • Fas Ligand Protein / metabolism*
  • Female
  • Hippocampus / metabolism
  • I-kappa B Proteins / metabolism
  • Ischemic Attack, Transient / metabolism*
  • NF-KappaB Inhibitor alpha
  • NF-kappa B / antagonists & inhibitors
  • NF-kappa B / metabolism
  • NFATC Transcription Factors / genetics
  • NFATC Transcription Factors / metabolism*
  • Neurons / metabolism
  • Promoter Regions, Genetic
  • Protein Binding
  • Random Allocation
  • Rats
  • Rats, Sprague-Dawley
  • Transcriptional Activation

Substances

  • Calcium Channel Blockers
  • Fas Ligand Protein
  • Faslg protein, rat
  • I-kappa B Proteins
  • NF-kappa B
  • NFATC Transcription Factors
  • Nfkbia protein, rat
  • NF-KappaB Inhibitor alpha
  • Cinnarizine
  • dipfluzine