Abstract
Currently, the origin of autoimmune diseases is considered to be multifactorial. Genetic predisposition, immune system malfunction or even backfire, hormonal regulation, and environmental factors, i.e. infections, all play important roles in the pathogenesis of autoimmune diseases such as the antiphospholipid syndrome (APS). New drugs and strategies aimed at preventing infections could further improve the outcome of APS and other autoimmune diseases.
MeSH terms
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Antibodies, Antineutrophil Cytoplasmic / immunology
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Antibodies, Antiphospholipid / immunology*
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Antigens, Bacterial / immunology
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Antigens, Viral / immunology
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Antiphospholipid Syndrome / etiology*
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Antiphospholipid Syndrome / physiopathology
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Autoantigens / immunology
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Autoimmunity
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DNA Virus Infections / complications*
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DNA Virus Infections / immunology
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DNA Viruses / immunology*
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DNA Viruses / pathogenicity
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GATA1 Transcription Factor / immunology
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Gram-Positive Bacteria / immunology*
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Gram-Positive Bacteria / pathogenicity
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Gram-Positive Bacterial Infections / complications*
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Gram-Positive Bacterial Infections / immunology
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Humans
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Immunity, Innate
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Molecular Mimicry
Substances
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Antibodies, Antineutrophil Cytoplasmic
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Antibodies, Antiphospholipid
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Antigens, Bacterial
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Antigens, Viral
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Autoantigens
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GATA1 Transcription Factor