Neuroinflammation as a neurotoxic mechanism in alcoholism: commentary on "Increased MCP-1 and microglia in various regions of human alcoholic brain"

Exp Neurol. 2008 Sep;213(1):10-7. doi: 10.1016/j.expneurol.2008.05.016. Epub 2008 Jul 14.
No abstract available

Publication types

  • Comment
  • Research Support, N.I.H., Extramural
  • Review

MeSH terms

  • Alcohol-Induced Disorders, Nervous System / immunology
  • Alcohol-Induced Disorders, Nervous System / pathology
  • Alcohol-Induced Disorders, Nervous System / physiopathology*
  • Brain / drug effects*
  • Brain / pathology
  • Brain / physiopathology
  • Central Nervous System Depressants / toxicity
  • Chemokine CCL2 / drug effects*
  • Chemokine CCL2 / immunology
  • Encephalitis / chemically induced*
  • Encephalitis / immunology
  • Encephalitis / physiopathology
  • Ethanol / toxicity
  • Gliosis / chemically induced
  • Gliosis / immunology
  • Gliosis / physiopathology
  • Microglia / drug effects*
  • Microglia / immunology
  • Nerve Degeneration / chemically induced
  • Nerve Degeneration / immunology
  • Nerve Degeneration / physiopathology

Substances

  • CCL2 protein, human
  • Central Nervous System Depressants
  • Chemokine CCL2
  • Ethanol