Abstract
Laboratory and population-based studies suggest that exposure to environmental toxicants may be one of several triggers for the development of endometriosis. We discuss evidence that modulation of the endometrial endocrine-immune interface could mechanistically link toxicant exposure to the development of this disease.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
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Review
MeSH terms
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Animals
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Dioxins / toxicity*
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Disease Models, Animal
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Endometriosis / chemically induced*
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Endometriosis / pathology
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Endometriosis / physiopathology
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Endometrium / physiology
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Environmental Exposure / adverse effects
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Female
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Humans
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Inflammation / chemically induced*
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Models, Biological