Novel role for osteopontin in cardiac fibrosis

Circ Res. 2008 Feb 15;102(3):270-2. doi: 10.1161/CIRCRESAHA.107.170555.
No abstract available

Publication types

  • Comment
  • Editorial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / biosynthesis
  • Actins / genetics
  • Animals
  • Cell Differentiation* / drug effects
  • Cells, Cultured
  • Collagen Type I / biosynthesis
  • Collagen Type I / genetics
  • Dermis / metabolism*
  • Dermis / pathology
  • Fibroblasts / metabolism*
  • Fibroblasts / pathology
  • Fibrosis
  • Focal Adhesions / genetics
  • Focal Adhesions / metabolism
  • Gene Expression Regulation / drug effects
  • HMGB1 Protein / biosynthesis
  • HMGB1 Protein / genetics
  • Heart Diseases / genetics
  • Heart Diseases / metabolism*
  • Heart Diseases / pathology
  • Humans
  • Mice
  • Mice, Knockout
  • Myocytes, Cardiac / metabolism*
  • Myocytes, Cardiac / pathology
  • Osteopontin / antagonists & inhibitors
  • Osteopontin / genetics
  • Osteopontin / metabolism*
  • Osteopontin / pharmacology
  • Pseudopodia / genetics
  • Pseudopodia / metabolism
  • RNA, Small Interfering / genetics
  • RNA, Small Interfering / pharmacology
  • Stress Fibers / metabolism
  • Transforming Growth Factor beta1 / metabolism
  • Transforming Growth Factor beta1 / pharmacology
  • Wound Healing / drug effects

Substances

  • Actins
  • Collagen Type I
  • HMGB1 Protein
  • RNA, Small Interfering
  • Spp1 protein, mouse
  • Transforming Growth Factor beta1
  • Osteopontin