Apoptosis and plaque destabilization in atherosclerosis: the role of macrophage apoptosis induced by cholesterol

Cell Death Differ. 2004 Jul:11 Suppl 1:S12-6. doi: 10.1038/sj.cdd.4401444.
No abstract available

Publication types

  • News
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Apoptosis / physiology*
  • Arterial Occlusive Diseases / etiology
  • Arteriosclerosis / complications
  • Arteriosclerosis / metabolism
  • Arteriosclerosis / physiopathology*
  • Caspases / metabolism
  • Cells, Cultured
  • Cholesterol / metabolism
  • Cholesterol / physiology*
  • Cholesterol Esters / metabolism
  • Cholesterol, LDL / metabolism
  • Endoplasmic Reticulum / metabolism
  • Endoplasmic Reticulum / physiology
  • Endothelium, Vascular / metabolism
  • Endothelium, Vascular / pathology
  • Endothelium, Vascular / physiopathology
  • Humans
  • Ketocholesterols / metabolism
  • Ketocholesterols / physiology
  • Lipoproteins, LDL / metabolism
  • Lipoproteins, LDL / physiology
  • Macrophages / metabolism*
  • Models, Biological
  • Protein Folding
  • Signal Transduction / physiology
  • Sterol O-Acyltransferase / metabolism
  • Sterol O-Acyltransferase / physiology

Substances

  • Cholesterol Esters
  • Cholesterol, LDL
  • Ketocholesterols
  • Lipoproteins, LDL
  • oxidized low density lipoprotein
  • Cholesterol
  • Sterol O-Acyltransferase
  • Caspases