Oxidant-induced iron signaling in Doxorubicin-mediated apoptosis

Methods Enzymol. 2004:378:362-82. doi: 10.1016/S0076-6879(04)78026-X.
No abstract available

Publication types

  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Aconitate Hydratase / analysis
  • Animals
  • Antibiotics, Antineoplastic / metabolism
  • Antibiotics, Antineoplastic / toxicity*
  • Antioxidants / pharmacology
  • Apoptosis / drug effects*
  • Caspases / metabolism
  • Cells, Cultured
  • Chelating Agents / pharmacology
  • Cytochromes c / metabolism
  • Doxorubicin / metabolism
  • Doxorubicin / toxicity*
  • Endothelium / drug effects
  • Endothelium / metabolism
  • Heart Diseases / chemically induced
  • Heart Diseases / drug therapy
  • Heart Diseases / physiopathology
  • Humans
  • Iron / metabolism*
  • Iron Chelating Agents / therapeutic use
  • Iron Regulatory Protein 1 / metabolism
  • Iron-Regulatory Proteins / metabolism
  • Mitochondria / enzymology
  • Mitochondria / metabolism
  • Muscle Cells / drug effects
  • Muscle Cells / metabolism
  • Oxidants / analysis
  • Oxidants / pharmacology*
  • Oxidation-Reduction
  • Oxidative Stress
  • RNA, Messenger / analysis
  • RNA, Messenger / metabolism
  • Razoxane / pharmacology
  • Reactive Oxygen Species / metabolism
  • Receptors, Transferrin / analysis
  • Receptors, Transferrin / drug effects
  • Receptors, Transferrin / metabolism
  • Response Elements
  • Signal Transduction*
  • Superoxides / analysis
  • Up-Regulation

Substances

  • Antibiotics, Antineoplastic
  • Antioxidants
  • Chelating Agents
  • Iron Chelating Agents
  • Iron-Regulatory Proteins
  • Oxidants
  • RNA, Messenger
  • Reactive Oxygen Species
  • Receptors, Transferrin
  • Superoxides
  • Razoxane
  • Doxorubicin
  • Cytochromes c
  • Iron
  • Caspases
  • Aconitate Hydratase
  • Iron Regulatory Protein 1