Cellular mechanism of vasoconstriction induced by angiotensin II: it remains to be determined

Circ Res. 2003 Nov 28;93(11):1015-7. doi: 10.1161/01.RES.0000105920.33926.60.
No abstract available

Publication types

  • Comment
  • Editorial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Angiotensin II / pharmacology
  • Angiotensin II / physiology*
  • Animals
  • Aorta, Abdominal / drug effects
  • Aorta, Abdominal / physiology
  • Calcium / metabolism
  • Femoral Artery / drug effects
  • Femoral Artery / physiology
  • GTP-Binding Protein alpha Subunits, Gq-G11 / metabolism
  • Humans
  • Mice
  • Mice, Knockout
  • Myosin Light Chains / metabolism
  • Myosin-Light-Chain Kinase / metabolism
  • Phosphorylation
  • Rabbits
  • Receptor, Angiotensin, Type 1 / deficiency
  • Receptor, Angiotensin, Type 1 / genetics
  • Receptor, Angiotensin, Type 1 / metabolism
  • Signal Transduction / physiology*
  • Time Factors
  • Type C Phospholipases / metabolism
  • Vasoconstriction / drug effects
  • Vasoconstriction / physiology*

Substances

  • Myosin Light Chains
  • Receptor, Angiotensin, Type 1
  • Angiotensin II
  • Myosin-Light-Chain Kinase
  • Type C Phospholipases
  • GTP-Binding Protein alpha Subunits, Gq-G11
  • Calcium