Hypoxia, HIFs, and cardiovascular development

Cold Spring Harb Symp Quant Biol. 2002:67:127-32. doi: 10.1101/sqb.2002.67.127.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cardiovascular System / embryology*
  • DNA-Binding Proteins / chemistry
  • DNA-Binding Proteins / deficiency
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / physiology*
  • Gene Expression Regulation, Developmental
  • Hematopoiesis
  • Humans
  • Hypoxia / physiopathology*
  • Hypoxia-Inducible Factor 1
  • Hypoxia-Inducible Factor 1, alpha Subunit
  • Mice
  • Mice, Knockout
  • Nuclear Proteins / chemistry
  • Nuclear Proteins / deficiency
  • Nuclear Proteins / genetics
  • Nuclear Proteins / physiology*
  • Oxygen / metabolism
  • Placenta / embryology
  • Signal Transduction
  • Transcription Factors*
  • von Hippel-Lindau Disease / embryology
  • von Hippel-Lindau Disease / genetics
  • von Hippel-Lindau Disease / physiopathology

Substances

  • DNA-Binding Proteins
  • HIF1A protein, human
  • Hif1a protein, mouse
  • Hypoxia-Inducible Factor 1
  • Hypoxia-Inducible Factor 1, alpha Subunit
  • Nuclear Proteins
  • Transcription Factors
  • Oxygen