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Table representation of search results timeline featuring number of search results per year.
Year | Number of Results |
---|---|
2014 | 1 |
2015 | 1 |
2019 | 1 |
2020 | 2 |
2021 | 1 |
2023 | 3 |
2024 | 0 |
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7 results
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Page 1
Reduced female fertility due to sequestration of RNA Pol II by pervasive transcription in exosome RNase-depleted oocytes.
Cell Rep. 2023 Oct 31;42(10):113247. doi: 10.1016/j.celrep.2023.113247. Epub 2023 Oct 12.
Cell Rep. 2023.
PMID: 37831603
Free article.
DIS3L2 ribonuclease degrades terminal-uridylated RNA to ensure oocyte maturation and female fertility.
Wu D, Pedroza M, Chang J, Dean J.
Wu D, et al.
Nucleic Acids Res. 2023 Apr 24;51(7):3078-3093. doi: 10.1093/nar/gkad061.
Nucleic Acids Res. 2023.
PMID: 36727488
Free PMC article.
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RNA exosome ribonuclease DIS3 degrades Pou6f1 to promote mouse pre-implantation cell differentiation.
Wu D, Dean J.
Wu D, et al.
Cell Rep. 2023 Feb 28;42(2):112047. doi: 10.1016/j.celrep.2023.112047. Epub 2023 Jan 30.
Cell Rep. 2023.
PMID: 36724075
Free PMC article.
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Sperm acrosome overgrowth and infertility in mice lacking chromosome 18 pachytene piRNA.
Choi H, Wang Z, Dean J.
Choi H, et al.
PLoS Genet. 2021 Apr 8;17(4):e1009485. doi: 10.1371/journal.pgen.1009485. eCollection 2021 Apr.
PLoS Genet. 2021.
PMID: 33831001
Free PMC article.
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Maternal factors regulating preimplantation development in mice.
Wu D, Dean J.
Wu D, et al.
Curr Top Dev Biol. 2020;140:317-340. doi: 10.1016/bs.ctdb.2019.10.006. Epub 2019 Nov 19.
Curr Top Dev Biol. 2020.
PMID: 32591079
Free PMC article.
Review.
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EXOSC10 sculpts the transcriptome during the growth-to-maturation transition in mouse oocytes.
Wu D, Dean J.
Wu D, et al.
Nucleic Acids Res. 2020 Jun 4;48(10):5349-5365. doi: 10.1093/nar/gkaa249.
Nucleic Acids Res. 2020.
PMID: 32313933
Free PMC article.
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Reprogramming the genome to totipotency in mouse embryos.
Zhou LQ, Dean J.
Zhou LQ, et al.
Trends Cell Biol. 2015 Feb;25(2):82-91. doi: 10.1016/j.tcb.2014.09.006. Epub 2014 Oct 21.
Trends Cell Biol. 2015.
PMID: 25448353
Free PMC article.
Review.
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