Rada C - Search Results

1

Apobec2 deficiency causes mitochondrial defects and mitophagy in skeletal muscle.

Sato Y, Ohtsubo H, Nihei N, Kaneko T, Sato Y, Adachi SI, Kondo S, Nakamura M, Mizunoya W, Iida H, Tatsumi R, Rada C, Yoshizawa F. Sato Y, et al. Among authors: rada c. FASEB J. 2018 Mar;32(3):1428-1439. doi: 10.1096/fj.201700493R. Epub 2018 Jan 3. FASEB J. 2018. PMID: 29127187 Free PMC article.

., Kondo, S., Nakamura, M., Mizunoya, W., Iida, H., Tatsumi, R., Rada, C., Yoshizawa, F. Apobec2 deficiency causes mitochondrial defects and mitophagy in skeletal muscle....

., Kondo, S., Nakamura, M., Mizunoya, W., Iida, H., Tatsumi, R., Rada, C., Yoshizawa, F. Apobec2 deficiency causes mitochondri …

2

Deficiency in APOBEC2 leads to a shift in muscle fiber type, diminished body mass, and myopathy.

Sato Y, Probst HC, Tatsumi R, Ikeuchi Y, Neuberger MS, Rada C. Sato Y, et al. Among authors: rada c. J Biol Chem. 2010 Mar 5;285(10):7111-8. doi: 10.1074/jbc.M109.052977. Epub 2009 Dec 17. J Biol Chem. 2010. PMID: 20022958 Free PMC article.

3

Mice deficient in APOBEC2 and APOBEC3.

Mikl MC, Watt IN, Lu M, Reik W, Davies SL, Neuberger MS, Rada C. Mikl MC, et al. Among authors: rada c. Mol Cell Biol. 2005 Aug;25(16):7270-7. doi: 10.1128/MCB.25.16.7270-7277.2005. Mol Cell Biol. 2005. PMID: 16055735 Free PMC article.

4

REG-γ associates with and modulates the abundance of nuclear activation-induced deaminase.

Uchimura Y, Barton LF, Rada C, Neuberger MS. Uchimura Y, et al. Among authors: rada c. J Exp Med. 2011 Nov 21;208(12):2385-91. doi: 10.1084/jem.20110856. Epub 2011 Oct 31. J Exp Med. 2011. PMID: 22042974 Free PMC article.

5

A high-throughput assay for DNA deaminases.

Wang M, Rada C, Neuberger MS. Wang M, et al. Among authors: rada c. Methods Mol Biol. 2011;718:171-84. doi: 10.1007/978-1-61779-018-8_11. Methods Mol Biol. 2011. PMID: 21370049

6

Altering the spectrum of immunoglobulin V gene somatic hypermutation by modifying the active site of AID.

Wang M, Rada C, Neuberger MS. Wang M, et al. Among authors: rada c. J Exp Med. 2010 Jan 18;207(1):141-53. doi: 10.1084/jem.20092238. Epub 2010 Jan 4. J Exp Med. 2010. PMID: 20048284 Free PMC article.

7

Deficiency in spliceosome-associated factor CTNNBL1 does not affect ongoing cell cycling but delays exit from quiescence and results in embryonic lethality in mice.

Chandra A, van Maldegem F, Andrews S, Neuberger MS, Rada C. Chandra A, et al. Among authors: rada c. Cell Cycle. 2013 Mar 1;12(5):732-42. doi: 10.4161/cc.23594. Epub 2013 Jan 23. Cell Cycle. 2013. PMID: 23343763 Free PMC article.

8

The in vivo pattern of AID targeting to immunoglobulin switch regions deduced from mutation spectra in msh2-/- ung-/- mice.

Xue K, Rada C, Neuberger MS. Xue K, et al. Among authors: rada c. J Exp Med. 2006 Sep 4;203(9):2085-94. doi: 10.1084/jem.20061067. Epub 2006 Aug 7. J Exp Med. 2006. PMID: 16894013 Free PMC article.

9

Cytoplasmic activation-induced cytidine deaminase (AID) exists in stoichiometric complex with translation elongation factor 1α (eEF1A).

Häsler J, Rada C, Neuberger MS. Häsler J, et al. Among authors: rada c. Proc Natl Acad Sci U S A. 2011 Nov 8;108(45):18366-71. doi: 10.1073/pnas.1106729108. Epub 2011 Oct 31. Proc Natl Acad Sci U S A. 2011. PMID: 22042842 Free PMC article.

10

Active RNAP pre-initiation sites are highly mutated by cytidine deaminases in yeast, with AID targeting small RNA genes.

Taylor BJ, Wu YL, Rada C. Taylor BJ, et al. Among authors: rada c. Elife. 2014 Sep 19;3:e03553. doi: 10.7554/eLife.03553. Elife. 2014. PMID: 25237741 Free PMC article.

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