Nguyen TH - Search Results

1

miR-122 promotes virus-induced lung disease by targeting SOCS1.

Collison AM, Sokulsky LA, Kepreotes E, Pereira de Siqueira A, Morten M, Edwards MR, Walton RP, Bartlett NW, Yang M, Nguyen TH, Johnston SL, Foster PS, Mattes J. Collison AM, et al. Among authors: nguyen th. JCI Insight. 2021 Apr 8;6(7):e127933. doi: 10.1172/jci.insight.127933. JCI Insight. 2021. PMID: 33830082 Free PMC article. Clinical Trial.

2

IL-17A is a common and critical driver of impaired lung function and immunopathology induced by influenza virus, rhinovirus and respiratory syncytial virus.

Liu X, Nguyen TH, Sokulsky L, Li X, Garcia Netto K, Hsu AC, Liu C, Laurie K, Barr I, Tay H, Eyers F, Foster PS, Yang M. Liu X, et al. Among authors: nguyen th. Respirology. 2021 Nov;26(11):1049-1059. doi: 10.1111/resp.14141. Epub 2021 Sep 1. Respirology. 2021. PMID: 34472161

3

TNF-α and Macrophages Are Critical for Respiratory Syncytial Virus-Induced Exacerbations in a Mouse Model of Allergic Airways Disease.

Nguyen TH, Maltby S, Simpson JL, Eyers F, Baines KJ, Gibson PG, Foster PS, Yang M. Nguyen TH, et al. J Immunol. 2016 May 1;196(9):3547-58. doi: 10.4049/jimmunol.1502339. Epub 2016 Apr 1. J Immunol. 2016. PMID: 27036916

4

Identification of IFN-γ and IL-27 as Critical Regulators of Respiratory Syncytial Virus-Induced Exacerbation of Allergic Airways Disease in a Mouse Model.

Nguyen TH, Maltby S, Tay HL, Eyers F, Foster PS, Yang M. Nguyen TH, et al. J Immunol. 2018 Jan 1;200(1):237-247. doi: 10.4049/jimmunol.1601950. Epub 2017 Nov 22. J Immunol. 2018. PMID: 29167232

5

A Critical Role for the CXCL3/CXCL5/CXCR2 Neutrophilic Chemotactic Axis in the Regulation of Type 2 Responses in a Model of Rhinoviral-Induced Asthma Exacerbation.

Sokulsky LA, Garcia-Netto K, Nguyen TH, Girkin JLN, Collison A, Mattes J, Kaiko G, Liu C, Bartlett NW, Yang M, Foster PS. Sokulsky LA, et al. Among authors: nguyen th. J Immunol. 2020 Nov 1;205(9):2468-2478. doi: 10.4049/jimmunol.1901350. Epub 2020 Sep 18. J Immunol. 2020. PMID: 32948685

6

MicroRNA-21 drives severe, steroid-insensitive experimental asthma by amplifying phosphoinositide 3-kinase-mediated suppression of histone deacetylase 2.

Kim RY, Horvat JC, Pinkerton JW, Starkey MR, Essilfie AT, Mayall JR, Nair PM, Hansbro NG, Jones B, Haw TJ, Sunkara KP, Nguyen TH, Jarnicki AG, Keely S, Mattes J, Adcock IM, Foster PS, Hansbro PM. Kim RY, et al. Among authors: nguyen th. J Allergy Clin Immunol. 2017 Feb;139(2):519-532. doi: 10.1016/j.jaci.2016.04.038. Epub 2016 Jun 10. J Allergy Clin Immunol. 2017. PMID: 27448447 Free article.

7

Bromodomain and Extra Terminal (BET) Inhibitor Suppresses Macrophage-Driven Steroid-Resistant Exacerbations of Airway Hyper-Responsiveness and Inflammation.

Nguyen TH, Maltby S, Eyers F, Foster PS, Yang M. Nguyen TH, et al. PLoS One. 2016 Sep 22;11(9):e0163392. doi: 10.1371/journal.pone.0163392. eCollection 2016. PLoS One. 2016. PMID: 27657907 Free PMC article.

8

Potential Role of MicroRNAs in the Regulation of Antiviral Responses to Influenza Infection.

Nguyen TH, Liu X, Su ZZ, Hsu AC, Foster PS, Yang M. Nguyen TH, et al. Front Immunol. 2018 Jul 4;9:1541. doi: 10.3389/fimmu.2018.01541. eCollection 2018. Front Immunol. 2018. PMID: 30022983 Free PMC article. Review.

9

Lipopolysaccharide induces steroid-resistant exacerbations in a mouse model of allergic airway disease collectively through IL-13 and pulmonary macrophage activation.

Hadjigol S, Netto KG, Maltby S, Tay HL, Nguyen TH, Hansbro NG, Eyers F, Hansbro PM, Yang M, Foster PS. Hadjigol S, et al. Among authors: nguyen th. Clin Exp Allergy. 2020 Jan;50(1):82-94. doi: 10.1111/cea.13505. Epub 2019 Oct 18. Clin Exp Allergy. 2020. PMID: 31579973

10

Ancestral sequence reconstruction of Mic60 reveals a residue signature supporting respiration in yeast.

Benning FMC, Bell TA, Nguyen TH, Syau D, Connell LB, daCosta CJB, Chao LH. Benning FMC, et al. Among authors: nguyen th. bioRxiv [Preprint]. 2024 Apr 29:2024.04.26.591372. doi: 10.1101/2024.04.26.591372. bioRxiv. 2024. PMID: 38746426 Free PMC article. Preprint.

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