Martínez-Cué C - Search Results

1

Down syndrome is an oxidative phosphorylation disorder.

Bayona-Bafaluy MP, Garrido-Pérez N, Meade P, Iglesias E, Jiménez-Salvador I, Montoya J, Martínez-Cué C, Ruiz-Pesini E. Bayona-Bafaluy MP, et al. Among authors: martinez cue c. Redox Biol. 2021 May;41:101871. doi: 10.1016/j.redox.2021.101871. Epub 2021 Jan 22. Redox Biol. 2021. PMID: 33540295 Free PMC article.

2

The Ts65Dn mouse model of Down syndrome shows reduced expression of the Bcl-X(L) antiapoptotic protein in the hippocampus not accompanied by changes in molecular or cellular markers of cell death.

Rueda N, Flórez J, Martínez-Cué C. Rueda N, et al. Int J Dev Neurosci. 2011 Nov;29(7):711-6. doi: 10.1016/j.ijdevneu.2011.06.001. Epub 2011 Jun 12. Int J Dev Neurosci. 2011. PMID: 21684326

3

Mouse models of Down syndrome as a tool to unravel the causes of mental disabilities.

Rueda N, Flórez J, Martínez-Cué C. Rueda N, et al. Neural Plast. 2012;2012:584071. doi: 10.1155/2012/584071. Epub 2012 May 22. Neural Plast. 2012. PMID: 22685678 Free PMC article. Review.

4

Apoptosis in Down's syndrome: lessons from studies of human and mouse models.

Rueda N, Flórez J, Martínez-Cué C. Rueda N, et al. Apoptosis. 2013 Feb;18(2):121-34. doi: 10.1007/s10495-012-0785-3. Apoptosis. 2013. PMID: 23224708

5

Reducing GABAA α5 receptor-mediated inhibition rescues functional and neuromorphological deficits in a mouse model of down syndrome.

Martínez-Cué C, Martínez P, Rueda N, Vidal R, García S, Vidal V, Corrales A, Montero JA, Pazos Á, Flórez J, Gasser R, Thomas AW, Honer M, Knoflach F, Trejo JL, Wettstein JG, Hernández MC. Martínez-Cué C, et al. J Neurosci. 2013 Feb 27;33(9):3953-66. doi: 10.1523/JNEUROSCI.1203-12.2013. J Neurosci. 2013. PMID: 23447605 Free PMC article.

6

Chronic melatonin treatment rescues electrophysiological and neuromorphological deficits in a mouse model of Down syndrome.

Corrales A, Vidal R, García S, Vidal V, Martínez P, García E, Flórez J, Sanchez-Barceló EJ, Martínez-Cué C, Rueda N. Corrales A, et al. J Pineal Res. 2014 Jan;56(1):51-61. doi: 10.1111/jpi.12097. Epub 2013 Nov 25. J Pineal Res. 2014. PMID: 24147912

7

Overexpression of Dyrk1A is implicated in several cognitive, electrophysiological and neuromorphological alterations found in a mouse model of Down syndrome.

García-Cerro S, Martínez P, Vidal V, Corrales A, Flórez J, Vidal R, Rueda N, Arbonés ML, Martínez-Cué C. García-Cerro S, et al. PLoS One. 2014 Sep 4;9(9):e106572. doi: 10.1371/journal.pone.0106572. eCollection 2014. PLoS One. 2014. PMID: 25188425 Free PMC article.

8

Chronic Melatonin Administration Reduced Oxidative Damage and Cellular Senescence in the Hippocampus of a Mouse Model of Down Syndrome.

Parisotto EB, Vidal V, García-Cerro S, Lantigua S, Wilhelm Filho D, Sanchez-Barceló EJ, Martínez-Cué C, Rueda N. Parisotto EB, et al. Neurochem Res. 2016 Nov;41(11):2904-2913. doi: 10.1007/s11064-016-2008-8. Epub 2016 Jul 23. Neurochem Res. 2016. PMID: 27450081

9

Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes.

García-Cerro S, Rueda N, Vidal V, Lantigua S, Martínez-Cué C. García-Cerro S, et al. Neurobiol Dis. 2017 Oct;106:76-88. doi: 10.1016/j.nbd.2017.06.010. Epub 2017 Jun 21. Neurobiol Dis. 2017. PMID: 28647555

10

Decreasing the Expression of GABA_A α5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome.

Vidal V, García-Cerro S, Martínez P, Corrales A, Lantigua S, Vidal R, Rueda N, Ozmen L, Hernández MC, Martínez-Cué C. Vidal V, et al. Mol Neurobiol. 2018 Jun;55(6):4745-4762. doi: 10.1007/s12035-017-0675-3. Epub 2017 Jul 17. Mol Neurobiol. 2018. PMID: 28717969

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