Krejci P - Search Results

1

An RNA aptamer restores defective bone growth in FGFR3-related skeletal dysplasia in mice.

Kimura T, Bosakova M, Nonaka Y, Hruba E, Yasuda K, Futakawa S, Kubota T, Fafilek B, Gregor T, Abraham SP, Gomolkova R, Belaskova S, Pesl M, Csukasi F, Duran I, Fujiwara M, Kavkova M, Zikmund T, Kaiser J, Buchtova M, Krakow D, Nakamura Y, Ozono K, Krejci P. Kimura T, et al. Among authors: krejci p. Sci Transl Med. 2021 May 5;13(592):eaba4226. doi: 10.1126/scitranslmed.aba4226. Sci Transl Med. 2021. PMID: 33952673

2

Interaction of fibroblast growth factor and C-natriuretic peptide signaling in regulation of chondrocyte proliferation and extracellular matrix homeostasis.

Krejci P, Masri B, Fontaine V, Mekikian PB, Weis M, Prats H, Wilcox WR. Krejci P, et al. J Cell Sci. 2005 Nov 1;118(Pt 21):5089-100. doi: 10.1242/jcs.02618. Epub 2005 Oct 18. J Cell Sci. 2005. PMID: 16234329

3

Fibroblast growth factors 1, 2, 17, and 19 are the predominant FGF ligands expressed in human fetal growth plate cartilage.

Krejci P, Krakow D, Mekikian PB, Wilcox WR. Krejci P, et al. Pediatr Res. 2007 Mar;61(3):267-72. doi: 10.1203/pdr.0b013e318030d157. Pediatr Res. 2007. PMID: 17314681

4

STAT1 and STAT3 do not participate in FGF-mediated growth arrest in chondrocytes.

Krejci P, Salazar L, Goodridge HS, Kashiwada TA, Schibler MJ, Jelinkova P, Thompson LM, Wilcox WR. Krejci P, et al. J Cell Sci. 2008 Feb 1;121(Pt 3):272-81. doi: 10.1242/jcs.017160. Epub 2008 Jan 15. J Cell Sci. 2008. PMID: 18198189

5

Analysis of STAT1 activation by six FGFR3 mutants associated with skeletal dysplasia undermines dominant role of STAT1 in FGFR3 signaling in cartilage.

Krejci P, Salazar L, Kashiwada TA, Chlebova K, Salasova A, Thompson LM, Bryja V, Kozubik A, Wilcox WR. Krejci P, et al. PLoS One. 2008;3(12):e3961. doi: 10.1371/journal.pone.0003961. Epub 2008 Dec 17. PLoS One. 2008. PMID: 19088846 Free PMC article.

6

FGFR3 signaling induces a reversible senescence phenotype in chondrocytes similar to oncogene-induced premature senescence.

Krejci P, Prochazkova J, Smutny J, Chlebova K, Lin P, Aklian A, Bryja V, Kozubik A, Wilcox WR. Krejci P, et al. Bone. 2010 Jul;47(1):102-10. doi: 10.1016/j.bone.2010.03.021. Epub 2010 Mar 31. Bone. 2010. PMID: 20362703 Free PMC article.

7

Bent bone dysplasia-FGFR2 type, a distinct skeletal disorder, has deficient canonical FGF signaling.

Merrill AE, Sarukhanov A, Krejci P, Idoni B, Camacho N, Estrada KD, Lyons KM, Deixler H, Robinson H, Chitayat D, Curry CJ, Lachman RS, Wilcox WR, Krakow D. Merrill AE, et al. Among authors: krejci p. Am J Hum Genet. 2012 Mar 9;90(3):550-7. doi: 10.1016/j.ajhg.2012.02.005. Epub 2012 Mar 1. Am J Hum Genet. 2012. PMID: 22387015 Free PMC article.

8

Exome sequencing identifies PDE4D mutations in acrodysostosis.

Lee H, Graham JM Jr, Rimoin DL, Lachman RS, Krejci P, Tompson SW, Nelson SF, Krakow D, Cohn DH. Lee H, et al. Among authors: krejci p. Am J Hum Genet. 2012 Apr 6;90(4):746-51. doi: 10.1016/j.ajhg.2012.03.004. Epub 2012 Mar 29. Am J Hum Genet. 2012. PMID: 22464252 Free PMC article.

9

The paradox of FGFR3 signaling in skeletal dysplasia: why chondrocytes growth arrest while other cells over proliferate.

Krejci P. Krejci P. Mutat Res Rev Mutat Res. 2014 Jan-Mar;759:40-8. doi: 10.1016/j.mrrev.2013.11.001. Epub 2013 Dec 1. Mutat Res Rev Mutat Res. 2014. PMID: 24295726 Review.

10

Effect of FGFR inhibitors on chicken limb development.

Horakova D, Cela P, Krejci P, Balek L, Moravcova Balkova S, Matalova E, Buchtova M. Horakova D, et al. Among authors: krejci p. Dev Growth Differ. 2014 Oct;56(8):555-72. doi: 10.1111/dgd.12156. Epub 2014 Oct 3. Dev Growth Differ. 2014. PMID: 25280231

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