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Table representation of search results timeline featuring number of search results per year.
Year | Number of Results |
---|---|
2020 | 1 |
2021 | 3 |
2023 | 2 |
2024 | 0 |
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5 results
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Page 1
The Cell-Autonomous Pro-Metastatic Activities of PD-L1 in Breast Cancer Are Regulated by N-Linked Glycosylation-Dependent Activation of STAT3 and STAT1.
Cells. 2023 Sep 23;12(19):2338. doi: 10.3390/cells12192338.
Cells. 2023.
PMID: 37830552
Free PMC article.
A Tumor Microenvironment-Driven Network Regulated by STAT3 and p65 Negatively Controls the Enrichment of Cancer Stem Cells in Human HR+/HER2- Breast Cancer.
Ben-Yaakov H, Meshel T, Pasmanik-Chor M, Körner C, Ben-Baruch A.
Ben-Yaakov H, et al.
Cancers (Basel). 2023 Apr 12;15(8):2255. doi: 10.3390/cancers15082255.
Cancers (Basel). 2023.
PMID: 37190183
Free PMC article.
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Continuous Inflammatory Stimulation Leads via Metabolic Plasticity to a Prometastatic Phenotype in Triple-Negative Breast Cancer Cells.
Morein D, Rubinstein-Achiasaf L, Brayer H, Dorot O, Pichinuk E, Ben-Yaakov H, Meshel T, Pasmanik-Chor M, Ben-Baruch A.
Morein D, et al. Among authors: ben yaakov h.
Cells. 2021 May 31;10(6):1356. doi: 10.3390/cells10061356.
Cells. 2021.
PMID: 34072893
Free PMC article.
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Persistent Inflammatory Stimulation Drives the Conversion of MSCs to Inflammatory CAFs That Promote Pro-Metastatic Characteristics in Breast Cancer Cells.
Rubinstein-Achiasaf L, Morein D, Ben-Yaakov H, Liubomirski Y, Meshel T, Elbaz E, Dorot O, Pichinuk E, Gershovits M, Weil M, Ben-Baruch A.
Rubinstein-Achiasaf L, et al. Among authors: ben yaakov h.
Cancers (Basel). 2021 Mar 23;13(6):1472. doi: 10.3390/cancers13061472.
Cancers (Basel). 2021.
PMID: 33806906
Free PMC article.
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Inflammation-Driven Breast Tumor Cell Plasticity: Stemness/EMT, Therapy Resistance and Dormancy.
Baram T, Rubinstein-Achiasaf L, Ben-Yaakov H, Ben-Baruch A.
Baram T, et al. Among authors: ben yaakov h.
Front Oncol. 2021 Jan 26;10:614468. doi: 10.3389/fonc.2020.614468. eCollection 2020.
Front Oncol. 2021.
PMID: 33585241
Free PMC article.
Review.
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