Friedman E - Search Results

1

Mirk kinase inhibition blocks the in vivo growth of pancreatic cancer cells.

Deng X, Friedman E. Deng X, et al. Among authors: friedman e. Genes Cancer. 2014 Sep;5(9-10):337-47. doi: 10.18632/genesandcancer.29. Genes Cancer. 2014. PMID: 25352950 Free PMC article.

2

Inactivation of mirk/dyrk1b kinase targets quiescent pancreatic cancer cells.

Ewton DZ, Hu J, Vilenchik M, Deng X, Luk KC, Polonskaia A, Hoffman AF, Zipf K, Boylan JF, Friedman EA. Ewton DZ, et al. Mol Cancer Ther. 2011 Nov;10(11):2104-14. doi: 10.1158/1535-7163.MCT-11-0498. Epub 2011 Aug 30. Mol Cancer Ther. 2011. PMID: 21878655 Free PMC article.

3

Rapid turnover of cell-cycle regulators found in Mirk/dyrk1B transfectants.

Ewton DZ, Lee K, Deng X, Lim S, Friedman E. Ewton DZ, et al. Among authors: friedman e. Int J Cancer. 2003 Jan 1;103(1):21-8. doi: 10.1002/ijc.10743. Int J Cancer. 2003. PMID: 12455049 Free article.

4

The kinase Mirk/Dyrk1B mediates cell survival in pancreatic ductal adenocarcinoma.

Deng X, Ewton DZ, Li S, Naqvi A, Mercer SE, Landas S, Friedman E. Deng X, et al. Among authors: friedman e. Cancer Res. 2006 Apr 15;66(8):4149-58. doi: 10.1158/0008-5472.CAN-05-3089. Cancer Res. 2006. PMID: 16618736

5

Mirk/Dyrk1B maintains the viability of quiescent pancreatic cancer cells by reducing levels of reactive oxygen species.

Deng X, Ewton DZ, Friedman E. Deng X, et al. Among authors: friedman e. Cancer Res. 2009 Apr 15;69(8):3317-24. doi: 10.1158/0008-5472.CAN-08-2903. Epub 2009 Apr 7. Cancer Res. 2009. PMID: 19351855 Free PMC article.

6

Mirk/dyrk1B kinase is upregulated following inhibition of mTOR.

Deng X, Hu J, Ewton DZ, Friedman E. Deng X, et al. Among authors: friedman e. Carcinogenesis. 2014 Sep;35(9):1968-76. doi: 10.1093/carcin/bgu058. Epub 2014 Mar 3. Carcinogenesis. 2014. PMID: 24590896 Free PMC article.

7

The normal function of the cancer kinase Mirk/dyrk1B is to reduce reactive oxygen species.

Deng X, Mercer SE, Sun CY, Friedman E. Deng X, et al. Among authors: friedman e. Genes Cancer. 2014 Apr;5(1-2):22-30. doi: 10.18632/genesandcancer.1. Genes Cancer. 2014. PMID: 24955215 Free PMC article.

8

Mirk kinase inhibition targets ovarian cancer ascites.

Deng X, Hu J, Cunningham MJ, Friedman E. Deng X, et al. Among authors: friedman e. Genes Cancer. 2014 May;5(5-6):201-11. doi: 10.18632/genesandcancer.19. Genes Cancer. 2014. PMID: 25061503 Free PMC article.

9

Mirk protein kinase is a mitogen-activated protein kinase substrate that mediates survival of colon cancer cells.

Lee K, Deng X, Friedman E. Lee K, et al. Among authors: friedman e. Cancer Res. 2000 Jul 1;60(13):3631-7. Cancer Res. 2000. PMID: 10910078

10

Transforming growth factor beta 1 induces proliferation in colon carcinoma cells by Ras-dependent, smad-independent down-regulation of p21cip1.

Yan Z, Kim GY, Deng X, Friedman E. Yan Z, et al. Among authors: friedman e. J Biol Chem. 2002 Mar 22;277(12):9870-9. doi: 10.1074/jbc.M107646200. Epub 2002 Jan 9. J Biol Chem. 2002. PMID: 11784716 Free article.

We recently demonstrated that colon carcinoma cells transfected with oncogenic cellular K-rasV12, but not oncogenic cellular H-rasV12, switched from TGFbeta1-insensitive to TGFbeta1-growth-stimulated and also became more invasive (Yan, Z., Deng, X., and Friedman, E. …

We recently demonstrated that colon carcinoma cells transfected with oncogenic cellular K-rasV12, but not oncogenic cellular H-rasV12, switc …

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