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Uncompensated mitochondrial oxidative stress underlies heart failure in an iPSC-derived model of congenital heart disease.
Xu X, Jin K, Bais AS, Zhu W, Yagi H, Feinstein TN, Nguyen PK, Criscione JD, Liu X, Beutner G, Karunakaran KB, Rao KS, He H, Adams P, Kuo CK, Kostka D, Pryhuber GS, Shiva S, Ganapathiraju MK, Porter GA Jr, Lin JI, Aronow B, Lo CW. Xu X, et al. Among authors: beutner g. Cell Stem Cell. 2022 May 5;29(5):840-855.e7. doi: 10.1016/j.stem.2022.03.003. Epub 2022 Apr 7. Cell Stem Cell. 2022. PMID: 35395180 Free PMC article.
An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore.
Alavian KN, Beutner G, Lazrove E, Sacchetti S, Park HA, Licznerski P, Li H, Nabili P, Hockensmith K, Graham M, Porter GA Jr, Jonas EA. Alavian KN, et al. Among authors: beutner g. Proc Natl Acad Sci U S A. 2014 Jul 22;111(29):10580-5. doi: 10.1073/pnas.1401591111. Epub 2014 Jun 16. Proc Natl Acad Sci U S A. 2014. PMID: 24979777 Free PMC article.
Physiological roles of the mitochondrial permeability transition pore.
Mnatsakanyan N, Beutner G, Porter GA, Alavian KN, Jonas EA. Mnatsakanyan N, et al. Among authors: beutner g. J Bioenerg Biomembr. 2017 Feb;49(1):13-25. doi: 10.1007/s10863-016-9652-1. Epub 2016 Feb 11. J Bioenerg Biomembr. 2017. PMID: 26868013 Free PMC article. Review.
68 results