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Table representation of search results timeline featuring number of search results per year.
Year | Number of Results |
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2017 | 1 |
2019 | 2 |
2020 | 1 |
2024 | 0 |
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Page 1
Glycolysis Is an Intrinsic Factor for Optimal Replication of a Norovirus.
mBio. 2019 Mar 12;10(2):e02175-18. doi: 10.1128/mBio.02175-18.
mBio. 2019.
PMID: 30862747
Free PMC article.
Norovirus-Mediated Modification of the Translational Landscape via Virus and Host-Induced Cleavage of Translation Initiation Factors.
Emmott E, Sorgeloos F, Caddy SL, Vashist S, Sosnovtsev S, Lloyd R, Heesom K, Locker N, Goodfellow I.
Emmott E, et al.
Mol Cell Proteomics. 2017 Apr;16(4 suppl 1):S215-S229. doi: 10.1074/mcp.M116.062448. Epub 2017 Jan 13.
Mol Cell Proteomics. 2017.
PMID: 28087593
Free PMC article.
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Noroviruses subvert the core stress granule component G3BP1 to promote viral VPg-dependent translation.
Hosmillo M, Lu J, McAllaster MR, Eaglesham JB, Wang X, Emmott E, Domingues P, Chaudhry Y, Fitzmaurice TJ, Tung MK, Panas MD, McInerney G, Locker N, Wilen CB, Goodfellow IG.
Hosmillo M, et al.
Elife. 2019 Aug 12;8:e46681. doi: 10.7554/eLife.46681.
Elife. 2019.
PMID: 31403400
Free PMC article.
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Norovirus infection results in eIF2α independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation.
Brocard M, Iadevaia V, Klein P, Hall B, Lewis G, Lu J, Burke J, Willcocks MM, Parker R, Goodfellow IG, Ruggieri A, Locker N.
Brocard M, et al.
PLoS Pathog. 2020 Jan 6;16(1):e1008250. doi: 10.1371/journal.ppat.1008250. eCollection 2020 Jan.
PLoS Pathog. 2020.
PMID: 31905230
Free PMC article.
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