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Page 1
Did you mean kao shan chen (8 results)?
Extracellular RNAs-TLR3 signaling contributes to cognitive impairment after chronic neuropathic pain in mice.
Zhang X, Gao R, Zhang C, Teng Y, Chen H, Li Q, Liu C, Wu J, Wei L, Deng L, Wu L, Ye-Lehmann S, Mao X, Liu J, Zhu T, Chen C. Zhang X, et al. Signal Transduct Target Ther. 2023 Aug 7;8(1):292. doi: 10.1038/s41392-023-01543-z. Signal Transduct Target Ther. 2023. PMID: 37544956 Free PMC article.
The level of TLR3 in hippocampal neurons increased in the chronic constriction injury (CCI) group than in the sham group in this study. Importantly, compared to the wild-type (WT) mice, TLR3 knockout (KO) mice and TLR3-specific neuronal knockdown mice both displayed …
The level of TLR3 in hippocampal neurons increased in the chronic constriction injury (CCI) group than in the sham group in this stud …
Melatonin receptor 1A, but not 1B, knockout decreases biliary damage and liver fibrosis during cholestatic liver injury.
Wu N, Carpino G, Ceci L, Baiocchi L, Francis H, Kennedy L, Zhou T, Chen L, Sato K, Kyritsi K, Meadows V, Ekser B, Franchitto A, Mancinelli R, Onori P, Gaudio E, Glaser S, Alpini G. Wu N, et al. Hepatology. 2022 Apr;75(4):797-813. doi: 10.1002/hep.32233. Epub 2021 Nov 24. Hepatology. 2022. PMID: 34743371 Free PMC article.
We aimed to determine the differential roles of MT1 and MT2 during cholestasis. APPROACH AND RESULTS: Wild-type (WT), MT1 knockout (KO), MT2KO, and MT1/MT2 double KO (DKO) mice underwent sham or bile duct ligation (BDL); these mice were also treated with mela …
We aimed to determine the differential roles of MT1 and MT2 during cholestasis. APPROACH AND RESULTS: Wild-type (WT), MT1 knockout (KO …
Extracellular RNAs-TLR3 signaling contributes to cognitive decline in a mouse model of postoperative cognitive dysfunction.
Chen C, Gao R, Li M, Wang Q, Chen H, Zhang S, Mao X, Behensky A, Zhang Z, Gan L, Li T, Liao R, Li Q, Yu H, Yang J, Zhu T, Liu J. Chen C, et al. Brain Behav Immun. 2019 Aug;80:439-451. doi: 10.1016/j.bbi.2019.04.024. Epub 2019 Apr 10. Brain Behav Immun. 2019. PMID: 30980952
Using a mouse model of POCD, 20-22 months wild-type (WT) mice were undergoing unilateral nephrectomy and increased TLR3 expression levels and co-localization with neuronal and microglial cells were found in the surgery group compared with the sham group. Compared with WT m …
Using a mouse model of POCD, 20-22 months wild-type (WT) mice were undergoing unilateral nephrectomy and increased TLR3 expression levels an …
The β-catenin/CBP signaling axis participates in sepsis-induced inflammatory lung injury.
Cheng X, Liu D, Ren X, Nie Y, Zhao Y, Chen R, Wang H. Cheng X, et al. Exp Biol Med (Maywood). 2022 Sep;247(17):1548-1557. doi: 10.1177/15353702221097316. Epub 2022 Jun 6. Exp Biol Med (Maywood). 2022. PMID: 35665630 Free PMC article.
C57BL/6 mice were randomly divided into five groups, namely, the sham, CLP, beta-catenin knockout (KO) + CLP, XAV-939 + CLP, and ICG-001 + CLP groups; the XAV-939 + CLP and ICG-001 + CLP groups were separately subjected to intraperitoneal injections of the beta-cate …
C57BL/6 mice were randomly divided into five groups, namely, the sham, CLP, beta-catenin knockout (KO) + CLP, XAV-939 + CLP, a …
Cathepsin B knockout confers significant brain protection in the mouse model of stroke.
Hu K, Park Y, Olivas Y, Chen A, Liu C, Hu B. Hu K, et al. Exp Neurol. 2023 Oct;368:114499. doi: 10.1016/j.expneurol.2023.114499. Epub 2023 Jul 26. Exp Neurol. 2023. PMID: 37506756
METHODS: A total of 57 mice were randomly assigned into four experimental groups: (i) wildtype (wt) sham control; (ii) wt middle cerebral artery occlusion (MCAO); (iii) cathepsin B (CTSB) knockout (KO) sham control; and (iv) CTSB KO MCAO. Mice were sub …
METHODS: A total of 57 mice were randomly assigned into four experimental groups: (i) wildtype (wt) sham control; (ii) wt middle cere …
Remote ischemic preconditioning protects against spinal cord ischemia-reperfusion injury in mice by activating NMDAR/AMPK/PGC-1α/SIRT3 signaling.
Gu C, Kong F, Zeng J, Geng X, Sun Y, Chen X. Gu C, et al. Cell Biosci. 2023 Mar 16;13(1):57. doi: 10.1186/s13578-023-00999-4. Cell Biosci. 2023. PMID: 36927808 Free PMC article.
BACKGROUND: To study the protective effects of delayed remote ischemic preconditioning (RIPC) against spinal cord ischemia-reperfusion injury (SCIRI) in mice and determine whether SIRT3 is involved in this protection and portrayed its upstream regulatory mechanisms. METHODS: In v …
BACKGROUND: To study the protective effects of delayed remote ischemic preconditioning (RIPC) against spinal cord ischemia-reperfusion injur …
Nox4 Expression Is Not Required for OVX-Induced Osteoblast Senescence and Bone Loss in Mice.
Chen JR, Lazarenko OP, Zhao H, Wankhade UD, Pedersen K, Watt J, Ronis MJJ. Chen JR, et al. JBMR Plus. 2020 Jul 23;4(8):e10376. doi: 10.1002/jbm4.10376. eCollection 2020 Aug. JBMR Plus. 2020. PMID: 32803108 Free PMC article.

Decreased bone mass including BMD and BMC were found in the OVX group compared with the sham control (p < 0.05); E2 treatment completely reversed OVX-induced bone loss. ...E2 blunted OVX-induced p53 and p21 overexpression, but not p16 and Nox4 in bone. In addition, 8- a

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Decreased bone mass including BMD and BMC were found in the OVX group compared with the sham control (p < 0.05); E2 treatment comp

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Sestrin2 protects the myocardium against radiation-induced damage.
Zeng YC, Chi F, Xing R, Zeng J, Gao S, Chen JJ, Wang HM, Duan QY, Sun YN, Niu N, Tang MY, Wu R. Zeng YC, et al. Radiat Environ Biophys. 2016 May;55(2):195-202. doi: 10.1007/s00411-016-0643-8. Epub 2016 Mar 15. Radiat Environ Biophys. 2016. PMID: 26980623
Mice with genetic deletion of the Sestrin2 (Sestrin2 knockout mice [Sestrin2 KO]) and treatment with irradiation (22 or 15 Gy) were used as independent approaches to determine the role of Sestrin2. ...After 22 or 15 Gy irradiation, the LV ejection fraction (EF) was impaire …
Mice with genetic deletion of the Sestrin2 (Sestrin2 knockout mice [Sestrin2 KO]) and treatment with irradiation (22 or 15 Gy) were u …
P53 Acetylation Exerts Critical Roles In Pressure Overload Induced Coronary Microvascular Dysfunction and Heart Failure.
He X, Cantrell AC, Williams QA, Gu W, Chen Y, Chen JX, Zeng H. He X, et al. bioRxiv [Preprint]. 2023 Feb 9:2023.02.08.527691. doi: 10.1101/2023.02.08.527691. bioRxiv. 2023. PMID: 36798200 Free PMC article. Updated. Preprint.
Additionally, the levels of SERCA-2 were significantly upregulated in sham p53(4KR) mice as well as in p53(4KR) mice after TAC. In vitro, p53(4KR) significantly improved endothelial cell (EC) glycolytic function and mitochondrial respiration, and enhanced EC proliferation …
Additionally, the levels of SERCA-2 were significantly upregulated in sham p53(4KR) mice as well as in p53(4KR) mice after TAC. In vi …
Sitagliptin protects rat kidneys from acute ischemia-reperfusion injury via upregulation of GLP-1 and GLP-1 receptors.
Chang MW, Chen CH, Chen YC, Wu YC, Zhen YY, Leu S, Tsai TH, Ko SF, Sung PH, Yang CC, Chiang HJ, Chang HW, Chen YT, Yip HK. Chang MW, et al. Acta Pharmacol Sin. 2015 Jan;36(1):119-30. doi: 10.1038/aps.2014.98. Epub 2014 Dec 15. Acta Pharmacol Sin. 2015. PMID: 25500876 Free PMC article. Review.
The aim of this study was to investigate whether sitagliptin protected the kidney function from acute ischemia-reperfusion (IR) injury in rats. METHODS: Adult male SD rats were categorized into 4 groups: sham control, IR injury, IR+sitagliptin (300 mg/kg) and IR+sitaglipti …
The aim of this study was to investigate whether sitagliptin protected the kidney function from acute ischemia-reperfusion (IR) injury in ra …
76 results