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Page 1
Showing results for tongji mi
Search for Tongxi Mi instead (1 results)
The interplay between sleep and gut microbiota.
Han M, Yuan S, Zhang J. Han M, et al. Brain Res Bull. 2022 Mar;180:131-146. doi: 10.1016/j.brainresbull.2021.12.016. Epub 2022 Jan 13. Brain Res Bull. 2022. PMID: 35032622 Review.
Myocardial Infarction with Nonobstructive Coronary Arteries (MINOCA): A Review of the Current Position.
Abdu FA, Mohammed AQ, Liu L, Xu Y, Che W. Abdu FA, et al. Cardiology. 2020;145(9):543-552. doi: 10.1159/000509100. Epub 2020 Aug 4. Cardiology. 2020. PMID: 32750696 Free article. Review.
Myocardial infarction with nonobstructive coronary arteries (MINOCA) remains a puzzling clinical entity that is characterized by clinical evidence of myocardial infarction (MI) with normal or near-normal coronary arteries on angiography (stenosis <50%). ...Since there a …
Myocardial infarction with nonobstructive coronary arteries (MINOCA) remains a puzzling clinical entity that is characterized by clinical ev …
Cardioprotective mechanism of SGLT2 inhibitor against myocardial infarction is through reduction of autosis.
Jiang K, Xu Y, Wang D, Chen F, Tu Z, Qian J, Xu S, Xu Y, Hwa J, Li J, Shang H, Xiang Y. Jiang K, et al. Protein Cell. 2022 May;13(5):336-359. doi: 10.1007/s13238-020-00809-4. Epub 2021 Jan 8. Protein Cell. 2022. PMID: 33417139 Free PMC article.
Here we demonstrated that the SGLT2 inhibitor, Empagliflozin (EMPA), suppresses cardiomyocytes autosis (autophagic cell death) to confer cardioprotective effects. Using myocardial infarction (MI) mouse models with and without diabetes mellitus, EMPA treatment significantly …
Here we demonstrated that the SGLT2 inhibitor, Empagliflozin (EMPA), suppresses cardiomyocytes autosis (autophagic cell death) to confer car …
LRP6 downregulation promotes cardiomyocyte proliferation and heart regeneration.
Wu Y, Zhou L, Liu H, Duan R, Zhou H, Zhang F, He X, Lu D, Xiong K, Xiong M, Zhuang J, Liu Y, Li L, Liang D, Chen YH. Wu Y, et al. Cell Res. 2021 Apr;31(4):450-462. doi: 10.1038/s41422-020-00411-7. Epub 2020 Sep 24. Cell Res. 2021. PMID: 32973339 Free PMC article.
Cardiomyocyte-specific deletion of Lrp6 in the mouse heart induced a robust regenerative response after myocardial infarction (MI), led to reduced MI area and improvement in left ventricular systolic function. In vivo genetic lineage tracing revealed that the newly …
Cardiomyocyte-specific deletion of Lrp6 in the mouse heart induced a robust regenerative response after myocardial infarction (MI), l …
Mycobacterium tuberculosis suppresses host DNA repair to boost its intracellular survival.
Liu S, Guan L, Peng C, Cheng Y, Cheng H, Wang F, Ma M, Zheng R, Ji Z, Cui P, Ren Y, Li L, Shi C, Wang J, Huang X, Cai X, Qu D, Zhang H, Mao Z, Liu H, Wang P, Sha W, Yang H, Wang L, Ge B. Liu S, et al. Cell Host Microbe. 2023 Nov 8;31(11):1820-1836.e10. doi: 10.1016/j.chom.2023.09.010. Epub 2023 Oct 16. Cell Host Microbe. 2023. PMID: 37848028 Free article.
TAK1 is an essential kinase for STING trafficking.
Ma M, Dang Y, Chang B, Wang F, Xu J, Chen L, Su H, Li J, Ge B, Chen C, Liu H. Ma M, et al. Mol Cell. 2023 Nov 2;83(21):3885-3903.e5. doi: 10.1016/j.molcel.2023.09.009. Epub 2023 Oct 12. Mol Cell. 2023. PMID: 37832545
Exercise downregulates HIPK2 and HIPK2 inhibition protects against myocardial infarction.
Zhou Q, Deng J, Yao J, Song J, Meng D, Zhu Y, Xu M, Liang Y, Xu J, Sluijter JP, Xiao J. Zhou Q, et al. EBioMedicine. 2021 Dec;74:103713. doi: 10.1016/j.ebiom.2021.103713. Epub 2021 Nov 24. EBioMedicine. 2021. PMID: 34837851 Free PMC article.
Furthermore, increasing levels of miR-222, targeting HIPK2, protected post-MI cardiac dysfunction, whereas cardiac dysfunction post-MI was aggravated in miR-222(-/-) rats. Moreover, serum miR-222 levels were significantly reduced in MI patients, as well as in …
Furthermore, increasing levels of miR-222, targeting HIPK2, protected post-MI cardiac dysfunction, whereas cardiac dysfunction post- …
TREM2 macrophage promotes cardiac repair in myocardial infarction by reprogramming metabolism via SLC25A53.
Gong S, Zhai M, Shi J, Yu G, Lei Z, Shi Y, Zeng Y, Ju P, Yang N, Zhang Z, Zhang D, Zhuang J, Yu Q, Zhang X, Jian W, Wang W, Peng W. Gong S, et al. Cell Death Differ. 2024 Feb;31(2):239-253. doi: 10.1038/s41418-023-01252-8. Epub 2024 Jan 5. Cell Death Differ. 2024. PMID: 38182899 Free PMC article.
Efferocytosis and metabolic reprogramming of macrophages play crucial roles in myocardial infarction (MI) repair. TREM2 has been proven to participate in phagocytosis and metabolism, but how it modulates myocardial infarction remains unclear. ...In summary, our study revea …
Efferocytosis and metabolic reprogramming of macrophages play crucial roles in myocardial infarction (MI) repair. TREM2 has been prov …
Host-mediated ubiquitination of a mycobacterial protein suppresses immunity.
Wang L, Wu J, Li J, Yang H, Tang T, Liang H, Zuo M, Wang J, Liu H, Liu F, Chen J, Liu Z, Wang Y, Peng C, Wu X, Zheng R, Huang X, Ran Y, Rao Z, Ge B. Wang L, et al. Nature. 2020 Jan;577(7792):682-688. doi: 10.1038/s41586-019-1915-7. Epub 2020 Jan 15. Nature. 2020. PMID: 31942069
4,533 results