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Year | Number of Results |
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2012 | 1 |
2013 | 1 |
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2021 | 1 |
2024 | 0 |
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Differentiation-dependent doxorubicin toxicity on H9c2 cardiomyoblasts.
Cardiovasc Toxicol. 2012 Dec;12(4):326-40. doi: 10.1007/s12012-012-9177-8.
Cardiovasc Toxicol. 2012.
PMID: 22744233
The Enhanced Efficacy of Intracellular Delivery of Doxorubicin/C6-Ceramide Combination Mediated by the F3 Peptide/Nucleolin System Is Supported by the Downregulation of the PI3K/Akt Pathway.
Cruz AF, Caleiras MB, Fonseca NA, Gonçalves N, Mendes VM, Sampaio SF, Moura V, Melo JB, Almeida RD, Manadas B, Simões S, Moreira JN.
Cruz AF, et al. Among authors: sampaio sf.
Cancers (Basel). 2021 Jun 18;13(12):3052. doi: 10.3390/cancers13123052.
Cancers (Basel). 2021.
PMID: 34207464
Free PMC article.
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Mitochondrial apoptosis-inducing factor is involved in doxorubicin-induced toxicity on H9c2 cardiomyoblasts.
Moreira AC, Branco AF, Sampaio SF, Cunha-Oliveira T, Martins TR, Holy J, Oliveira PJ, Sardão VA.
Moreira AC, et al. Among authors: sampaio sf.
Biochim Biophys Acta. 2014 Dec;1842(12 Pt A):2468-78. doi: 10.1016/j.bbadis.2014.09.015. Epub 2014 Oct 2.
Biochim Biophys Acta. 2014.
PMID: 25283819
Free article.
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p66Shc signaling is involved in stress responses elicited by anthracycline treatment of rat cardiomyoblasts.
Sampaio SF, Branco AF, Wojtala A, Vega-Naredo I, Wieckowski MR, Oliveira PJ.
Sampaio SF, et al.
Arch Toxicol. 2016 Jul;90(7):1669-84. doi: 10.1007/s00204-015-1583-9. Epub 2015 Aug 30.
Arch Toxicol. 2016.
PMID: 26318906
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Mitochondrial disruption occurs downstream from β-adrenergic overactivation by isoproterenol in differentiated, but not undifferentiated H9c2 cardiomyoblasts: differential activation of stress and survival pathways.
Branco AF, Sampaio SF, Wieckowski MR, Sardão VA, Oliveira PJ.
Branco AF, et al. Among authors: sampaio sf.
Int J Biochem Cell Biol. 2013 Nov;45(11):2379-91. doi: 10.1016/j.biocel.2013.08.006. Epub 2013 Aug 16.
Int J Biochem Cell Biol. 2013.
PMID: 23958426
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