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2005 | 3 |
2007 | 1 |
2024 | 0 |
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Diesel exhaust particles induce the over expression of tumor necrosis factor-alpha (TNF-alpha) gene in alveolar macrophages and failed to induce apoptosis through activation of nuclear factor-kappaB (NF-kappaB).
Int J Environ Res Public Health. 2005 Apr;2(1):107-13. doi: 10.3390/ijerph2005010107.
Int J Environ Res Public Health. 2005.
PMID: 16705808
Free PMC article.
Activation of transcription factor IL-6 (NF-IL-6) and nuclear factor-kappaB (NF-kappaB) by lipid ozonation products is crucial to interleukin-8 gene expression in human airway epithelial cells.
Kafoury RM, Hernandez JM, Lasky JA, Toscano WA Jr, Friedman M.
Kafoury RM, et al.
Environ Toxicol. 2007 Apr;22(2):159-68. doi: 10.1002/tox.20246.
Environ Toxicol. 2007.
PMID: 17366569
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Application of quantitative structure activity relationship (QSAR) models to predict ozone toxicity in the lung.
Kafoury RM, Huang MJ.
Kafoury RM, et al.
Environ Toxicol. 2005 Aug;20(4):441-8. doi: 10.1002/tox.20130.
Environ Toxicol. 2005.
PMID: 16007646
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Ozone enhances diesel exhaust particles (DEP)-induced interleukin-8 (IL-8) gene expression in human airway epithelial cells through activation of nuclear factors- kappaB (NF-kappaB) and IL-6 (NF-IL6).
Kafoury RM, Kelley J.
Kafoury RM, et al.
Int J Environ Res Public Health. 2005 Dec;2(3-4):403-10. doi: 10.3390/ijerph2005030004.
Int J Environ Res Public Health. 2005.
PMID: 16819095
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