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Page 1
Hereditary Tyrosinemia Type 1 Mice under Continuous Nitisinone Treatment Display Remnants of an Uncorrected Liver Disease Phenotype.
Genes (Basel). 2023 Mar 11;14(3):693. doi: 10.3390/genes14030693.
Genes (Basel). 2023.
PMID: 36980965
Free PMC article.
Short-term nitisinone discontinuation of hereditary tyrosinemia type 1 mice causes metabolic alterations in glutathione metabolism/biosynthesis and multiple amino acid degradation pathways.
Haaike CV, Brendan NP, Sven VL, Andrew DS, Lionel M, Georges C, Philippe G, Paul C, Dimitri B, Geert M, Rao RL, Tamara V, James A G, Joery K.
Haaike CV, et al. Among authors: paul c.
Genes Dis. 2022 Dec 26;10(5):1759-1762. doi: 10.1016/j.gendis.2022.11.013. eCollection 2023 Sep.
Genes Dis. 2022.
PMID: 37492745
Free PMC article.
No abstract available.
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Oxidative Stress, Glutathione Metabolism, and Liver Regeneration Pathways Are Activated in Hereditary Tyrosinemia Type 1 Mice upon Short-Term Nitisinone Discontinuation.
Colemonts-Vroninks H, Neuckermans J, Marcelis L, Claes P, Branson S, Casimir G, Goyens P, Martens GA, Vanhaecke T, De Kock J.
Colemonts-Vroninks H, et al. Among authors: claes p.
Genes (Basel). 2020 Dec 22;12(1):3. doi: 10.3390/genes12010003.
Genes (Basel). 2020.
PMID: 33375092
Free PMC article.
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Simple and quick method for whole-liver decellularization: a novel in vitro three-dimensional bioengineering tool?
De Kock J, Ceelen L, De Spiegelaere W, Casteleyn C, Claes P, Vanhaecke T, Rogiers V.
De Kock J, et al. Among authors: claes p.
Arch Toxicol. 2011 Jun;85(6):607-12. doi: 10.1007/s00204-011-0706-1. Epub 2011 Apr 22.
Arch Toxicol. 2011.
PMID: 21512802
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