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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1951 1
1957 2
1961 1
1964 2
1965 5
1966 2
1968 1
1973 1
1974 2
1975 21
1976 27
1977 22
1978 33
1979 39
1980 35
1981 46
1982 45
1983 55
1984 36
1985 52
1986 51
1987 74
1988 70
1989 79
1990 93
1991 85
1992 89
1993 100
1994 83
1995 128
1996 128
1997 136
1998 150
1999 144
2000 194
2001 145
2002 146
2003 164
2004 193
2005 209
2006 155
2007 157
2008 195
2009 192
2010 204
2011 250
2012 280
2013 300
2014 346
2015 351
2016 335
2017 331
2018 337
2019 369
2020 424
2021 449
2022 421
2023 374
2024 141

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7,618 results

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Page 1
Lambert-Eaton Myasthenic Syndrome.
Kesner VG, Oh SJ, Dimachkie MM, Barohn RJ. Kesner VG, et al. Neurol Clin. 2018 May;36(2):379-394. doi: 10.1016/j.ncl.2018.01.008. Neurol Clin. 2018. PMID: 29655456 Free PMC article. Review.
Lambert-Eaton myasthenic syndrome is a paraneoplastic or primary autoimmune neuromuscular junction disorder characterized by proximal weakness, autonomic dysfunction and ariflexia. ...More than half of Lambert-Eaton myasthenic syndrome cases are associated with smal
Lambert-Eaton myasthenic syndrome is a paraneoplastic or primary autoimmune neuromuscular junction disorder characterized by proximal
Lambert-Eaton myasthenic syndrome.
Lipka AF, Verschuuren JJGM. Lipka AF, et al. Handb Clin Neurol. 2024;200:307-325. doi: 10.1016/B978-0-12-823912-4.00012-8. Handb Clin Neurol. 2024. PMID: 38494285 Review.
Lambert-Eaton myasthenic syndrome (LEMS) is a rare autoimmune disease characterized by proximal muscle weakness, loss of tendon reflexes, and autonomic dysfunction. Muscle weakness usually starts in the upper legs and can progress to oculobulbar and in severe cases respira
Lambert-Eaton myasthenic syndrome (LEMS) is a rare autoimmune disease characterized by proximal muscle weakness, loss of tendon refle
Lambert-Eaton syndrome.
Pascuzzi RM, Kim YI. Pascuzzi RM, et al. Semin Neurol. 1990 Mar;10(1):35-41. doi: 10.1055/s-2008-1041251. Semin Neurol. 1990. PMID: 2189179 Review.
Lambert-Eaton myasthenic syndrome.
Newsom-Davis J. Newsom-Davis J. Rev Neurol (Paris). 2004 Feb;160(2):177-80. doi: 10.1016/s0035-3787(04)70888-7. Rev Neurol (Paris). 2004. PMID: 15034474 Review.
The disorder is paraneoplastic (small cell lung cancer) in about 60p. cent (P-LEMS); no cancer is associated in the remainder (NP-LEMS). LEMS affects all races. NP-LEMS can occur in childhood as well as adult life; P-LEMS is unusual at<30 Years. The weakness resu …
The disorder is paraneoplastic (small cell lung cancer) in about 60p. cent (P-LEMS); no cancer is associated in the remainder (NP-LEM …
[Lambert-Eaton myasthenic syndrome].
Hernández MA, Köhler AA, Marrodán M, Lautre A, Brand P, Nogués M, Barroso F. Hernández MA, et al. Rev Neurol. 2021 Aug 1;73(3):96-100. doi: 10.33588/rn.7303.2021140. Rev Neurol. 2021. PMID: 34291446 Free article. Spanish.
RESULTS: Four patients were diagnosed with T-LEMS, two of them with small-cell lung carcinoma. Of the nine patients with NT-LEMS, five had a DELTA-P score of 3 and 4. Nine patients presented with the classic clinical triad from the onset of the disease. ...CONCLUSIONS: The …
RESULTS: Four patients were diagnosed with T-LEMS, two of them with small-cell lung carcinoma. Of the nine patients with NT-LEMS, five had a …
Lambert-Eaton myasthenic syndrome.
Seneviratne U, de Silva R. Seneviratne U, et al. Postgrad Med J. 1999 Sep;75(887):516-20. doi: 10.1136/pgmj.75.887.516. Postgrad Med J. 1999. PMID: 10616683 Free PMC article. Review.
The Lambert-Eaton myasthenic syndrome is a neuromuscular disorder characterised by defective neurotransmitter release at autonomic neurones and presynaptic terminals of the neuromuscular junction. ...Assay of VGCC antibody titres and electrophysiological tests help to diff …
The Lambert-Eaton myasthenic syndrome is a neuromuscular disorder characterised by defective neurotransmitter release at autonomic ne …
Lambert-Eaton myasthenic syndrome.
Mareska M, Gutmann L. Mareska M, et al. Semin Neurol. 2004 Jun;24(2):149-53. doi: 10.1055/s-2004-830900. Semin Neurol. 2004. PMID: 15257511 Review.
Lambert-Eaton myasthenic syndrome (LEMS) is an idiopathic or paraneoplastic syndrome producing antibodies against presynaptic voltage-gated P/Q calcium channels. ...
Lambert-Eaton myasthenic syndrome (LEMS) is an idiopathic or paraneoplastic syndrome producing antibodies against presynaptic voltage
[Autoantibody against the presynaptic P/Q-type voltage-gated calcium channel in Lambert-Eaton myasthenic syndrome].
Sakai W, Nakane S, Matsuo H. Sakai W, et al. Brain Nerve. 2013 Apr;65(4):441-8. Brain Nerve. 2013. PMID: 23568992 Review. Japanese.
Since then, other neurological disorders associated with autoantibodies have been identified, each associated with an antibody against a ligand- or voltage-gated ion channel. Autoantibodies against P/Q-type voltage-gated calcium channels (VGCCs) are detected in patients wi …
Since then, other neurological disorders associated with autoantibodies have been identified, each associated with an antibody against a lig …
Asymptomatic Lambert-Eaton syndrome.
Denys EH, Lennon VA. Denys EH, et al. Muscle Nerve. 2014 May;49(5):764-7. doi: 10.1002/mus.24126. Epub 2014 Apr 8. Muscle Nerve. 2014. PMID: 24259319
INTRODUCTION: All descriptions of the Lambert-Eaton syndrome are based on symptomatic patients. We incidentally encountered electromyographic evidence of this syndrome in a patient being studied for another reason. ...Multiple IgG markers of thyrogastric autoimmunity also …
INTRODUCTION: All descriptions of the Lambert-Eaton syndrome are based on symptomatic patients. We incidentally encountered electromy …
Synaptic Pathophysiology and Treatment of Lambert-Eaton Myasthenic Syndrome.
Tarr TB, Wipf P, Meriney SD. Tarr TB, et al. Mol Neurobiol. 2015 Aug;52(1):456-63. doi: 10.1007/s12035-014-8887-2. Epub 2014 Sep 9. Mol Neurobiol. 2015. PMID: 25195700 Free PMC article. Review.
Lambert-Eaton myasthenic syndrome (LEMS) is an autoimmune disease that disrupts the normally reliable neurotransmission at the neuromuscular junction (NMJ). This disruption is thought to result from an autoantibody-mediated removal of a subset of the P/Q-type Ca(2+)
Lambert-Eaton myasthenic syndrome (LEMS) is an autoimmune disease that disrupts the normally reliable neurotransmission at the neurom
7,618 results