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Page 1
Neuropathic and cAMP-induced pain behavior is ameliorated in mice lacking CNGB1.
Neuropharmacology. 2020 Jul;171:108087. doi: 10.1016/j.neuropharm.2020.108087. Epub 2020 Apr 6.
Neuropharmacology. 2020.
PMID: 32272140
Rab7-a novel redox target that modulates inflammatory pain processing.
Kallenborn-Gerhardt W, Möser CV, Lorenz JE, Steger M, Heidler J, Scheving R, Petersen J, Kennel L, Flauaus C, Lu R, Edinger AL, Tegeder I, Geisslinger G, Heide H, Wittig I, Schmidtko A.
Kallenborn-Gerhardt W, et al. Among authors: kennel l.
Pain. 2017 Jul;158(7):1354-1365. doi: 10.1097/j.pain.0000000000000920.
Pain. 2017.
PMID: 28394828
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KCa3.1 channels modulate the processing of noxious chemical stimuli in mice.
Lu R, Flauaus C, Kennel L, Petersen J, Drees O, Kallenborn-Gerhardt W, Ruth P, Lukowski R, Schmidtko A.
Lu R, et al. Among authors: kennel l.
Neuropharmacology. 2017 Oct;125:386-395. doi: 10.1016/j.neuropharm.2017.08.021. Epub 2017 Aug 18.
Neuropharmacology. 2017.
PMID: 28823609
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Distinct functions of soluble guanylyl cyclase isoforms NO-GC1 and NO-GC2 in inflammatory and neuropathic pain processing.
Petersen J, Mergia E, Kennel L, Drees O, Steubing RD, Real CI, Kallenborn-Gerhardt W, Lu R, Friebe A, Koesling D, Schmidtko A.
Petersen J, et al. Among authors: kennel l.
Pain. 2019 Mar;160(3):607-618. doi: 10.1097/j.pain.0000000000001440.
Pain. 2019.
PMID: 30422870
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cGMP Imaging in Brain Slices Reveals Brain Region-Specific Activity of NO-Sensitive Guanylyl Cyclases (NO-GCs) and NO-GC Stimulators.
Peters S, Paolillo M, Mergia E, Koesling D, Kennel L, Schmidtko A, Russwurm M, Feil R.
Peters S, et al. Among authors: kennel l.
Int J Mol Sci. 2018 Aug 7;19(8):2313. doi: 10.3390/ijms19082313.
Int J Mol Sci. 2018.
PMID: 30087260
Free PMC article.
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