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Page 1
Plasminogen alleles influence susceptibility to invasive aspergillosis.
Zaas AK, Liao G, Chien JW, Weinberg C, Shore D, Giles SS, Marr KA, Usuka J, Burch LH, Perera L, Perfect JR, Peltz G, Schwartz DA. Zaas AK, et al. PLoS Genet. 2008 Jun 20;4(6):e1000101. doi: 10.1371/journal.pgen.1000101. PLoS Genet. 2008. PMID: 18566672 Free PMC article.
Fatal and Nonfatal Events Within 14 days After Early, Intensive Mobilization Poststroke.
Bernhardt J, Borschmann K, Collier JM, Thrift AG, Langhorne P, Middleton S, Lindley RI, Dewey HM, Bath P, Said CM, Churilov L, Ellery F, Bladin C, Reid CM, Frayne JH, Srikanth V, Read SJ, Donnan GA; AVERT Trialists Collaboration Group. Bernhardt J, et al. Neurology. 2021 Feb 22;96(8):e1156-e1166. doi: 10.1212/WNL.0000000000011106. Neurology. 2021. PMID: 33144512 Free PMC article.
Prespecified dose-response analysis for A Very Early Rehabilitation Trial (AVERT).
Bernhardt J, Churilov L, Ellery F, Collier J, Chamberlain J, Langhorne P, Lindley RI, Moodie M, Dewey H, Thrift AG, Donnan G; AVERT Collaboration Group. Bernhardt J, et al. Neurology. 2016 Jun 7;86(23):2138-45. doi: 10.1212/WNL.0000000000002459. Epub 2016 Feb 17. Neurology. 2016. PMID: 26888985 Free PMC article. Clinical Trial.
A global-scale screening of non-native aquatic organisms to identify potentially invasive species under current and future climate conditions.
Vilizzi L, Copp GH, Hill JE, Adamovich B, Aislabie L, Akin D, Al-Faisal AJ, Almeida D, Azmai MNA, Bakiu R, Bellati A, Bernier R, Bies JM, Bilge G, Branco P, Bui TD, Canning-Clode J, Cardoso Ramos HA, Castellanos-Galindo GA, Castro N, Chaichana R, Chainho P, Chan J, Cunico AM, Curd A, Dangchana P, Dashinov D, Davison PI, de Camargo MP, Dodd JA, Durland Donahou AL, Edsman L, Ekmekçi FG, Elphinstone-Davis J, Erős T, Evangelista C, Fenwick G, Ferincz Á, Ferreira T, Feunteun E, Filiz H, Forneck SC, Gajduchenko HS, Gama Monteiro J, Gestoso I, Giannetto D, Gilles AS Jr, Gizzi F, Glamuzina B, Glamuzina L, Goldsmit J, Gollasch S, Goulletquer P, Grabowska J, Harmer R, Haubrock PJ, He D, Hean JW, Herczeg G, Howland KL, İlhan A, Interesova E, Jakubčinová K, Jelmert A, Johnsen SI, Kakareko T, Kanongdate K, Killi N, Kim JE, Kırankaya ŞG, Kňazovická D, Kopecký O, Kostov V, Koutsikos N, Kozic S, Kuljanishvili T, Kumar B, Kumar L, Kurita Y, Kurtul I, Lazzaro L, Lee L, Lehtiniemi M, Leonardi G, Leuven RSEW, Li S, Lipinskaya T, Liu F, Lloyd L, Lorenzoni M, Luna SA, Lyons TJ, Magellan K, Malmstrøm M, Marchini A, Marr SM, Masson G, Masson L, McKenzie CH, Memedemin D, Mendoza R, Minchin D, Miossec L, M… See abstract for full author list ➔ Vilizzi L, et al. Sci Total Environ. 2021 Sep 20;788:147868. doi: 10.1016/j.scitotenv.2021.147868. Epub 2021 May 20. Sci Total Environ. 2021. PMID: 34134389 Free article.
Functional consequences of substitution mutations in MepR, a repressor of the Staphylococcus aureus MepA multidrug efflux pump gene.
Schindler BD, Seo SM, Jacinto PL, Kumaraswami M, Birukou I, Brennan RG, Kaatz GW. Schindler BD, et al. J Bacteriol. 2013 Aug;195(16):3651-62. doi: 10.1128/JB.00565-13. Epub 2013 Jun 7. J Bacteriol. 2013. PMID: 23749979 Free PMC article.
The expression of mepA, encoding the Staphylococcus aureus MepA multidrug efflux protein, is repressed by the MarR homologue MepR. MepR dimers bind differently to operators upstream of mepR and mepA, with affinity being greatest at the mepA operator. ...
The expression of mepA, encoding the Staphylococcus aureus MepA multidrug efflux protein, is repressed by the MarR homologue MepR. Me …
Structural mechanism of transcription regulation of the Staphylococcus aureus multidrug efflux operon mepRA by the MarR family repressor MepR.
Birukou I, Seo SM, Schindler BD, Kaatz GW, Brennan RG. Birukou I, et al. Nucleic Acids Res. 2014 Feb;42(4):2774-88. doi: 10.1093/nar/gkt1215. Epub 2013 Nov 28. Nucleic Acids Res. 2014. PMID: 24293644 Free PMC article.
MepR, a member of the multiple antibiotic resistance regulator (MarR) family, represses mepA and its own gene. Here, we report the structure of a MepR-mepR operator complex. ...The wings insert into the flanking minor grooves, whereby residue Arg87, buttressed by Asp85, in …
MepR, a member of the multiple antibiotic resistance regulator (MarR) family, represses mepA and its own gene. Here, we report the st …
Multidrug resistance in Staphylococcus aureus due to overexpression of a novel multidrug and toxin extrusion (MATE) transport protein.
Kaatz GW, McAleese F, Seo SM. Kaatz GW, et al. Antimicrob Agents Chemother. 2005 May;49(5):1857-64. doi: 10.1128/AAC.49.5.1857-1864.2005. Antimicrob Agents Chemother. 2005. PMID: 15855507 Free PMC article.
There was concomitant overexpression of ORFs in close proximity to mepA (approximately 100 bp) encoding a MarR-type regulator (mepR, upstream of mepA) and a protein of unknown function (mepB, downstream). ...
There was concomitant overexpression of ORFs in close proximity to mepA (approximately 100 bp) encoding a MarR-type regulator (mepR, …
MepR, a repressor of the Staphylococcus aureus MATE family multidrug efflux pump MepA, is a substrate-responsive regulatory protein.
Kaatz GW, DeMarco CE, Seo SM. Kaatz GW, et al. Antimicrob Agents Chemother. 2006 Apr;50(4):1276-81. doi: 10.1128/AAC.50.4.1276-1281.2006. Antimicrob Agents Chemother. 2006. PMID: 16569840 Free PMC article.
The mepRAB gene cluster of Staphylococcus aureus encodes a MarR family repressor (MepR; known to repress mepA expression), a MATE family multidrug efflux pump (MepA), and a protein of unknown function (MepB). ...
The mepRAB gene cluster of Staphylococcus aureus encodes a MarR family repressor (MepR; known to repress mepA expression), a MATE fam …
Exploring threats to generalisability in a large international rehabilitation trial (AVERT).
Bernhardt J, Raffelt A, Churilov L, Lindley RI, Speare S, Ancliffe J, Katijjahbe MA, Hameed S, Lennon S, McRae A, Tan D, Quiney J, Williamson HC, Collier J, Dewey HM, Donnan GA, Langhorne P, Thrift AG; AVERT Trialists’ Collaboration. Bernhardt J, et al. BMJ Open. 2015 Aug 17;5(8):e008378. doi: 10.1136/bmjopen-2015-008378. BMJ Open. 2015. PMID: 26283667 Free PMC article. Clinical Trial.
The most common reason for non-recruitment was late arrival to hospital (ie, >24 h). Overall, being older and female reduced the odds of recruitment to the trial. ...
The most common reason for non-recruitment was late arrival to hospital (ie, >24 h). Overall, being older and female reduced the o …