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indrak vitro
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Indrek Virro
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A new alpha-thalassemia-2 deletion resulting in microcytosis and hypochromia and in vitro chain imbalance in the heterozygote.
Am J Hematol. 1993 Jun;43(2):144-5. doi: 10.1002/ajh.2830430214.
Am J Hematol. 1993.
PMID: 8342542
Unexpectedly, the heterozygote had a mild anemia with a marked microcytosis and hypochromia, and an in vitro alpha/beta chain synthesis ratio of 0.62-0.66. It is suggested that the deletion includes a sequence that is involved in the in cis regulation of the alpha 2-globin …
Unexpectedly, the heterozygote had a mild anemia with a marked microcytosis and hypochromia, and an in vitro alpha/beta chain synthes …
Flow cytometric monitoring of the in vitro inhibition of the phosphorylation of CRKL and of SRC family kinases in patients with chronic myelogenous leukemia treated with tyrosine kinase inhibitors.
Krupkova L, Mojzikova R, Novotny J, Gazdova J, Divoka M, Skoumalova I, Rohon P, Jarosova M, Indrak K, Faber E, Divoky V.
Krupkova L, et al.
Int J Lab Hematol. 2015 Feb;37(1):e11-5. doi: 10.1111/ijlh.12258. Epub 2014 May 21.
Int J Lab Hematol. 2015.
PMID: 24845482
No abstract available.
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Identification of e6a2 BCR-ABL fusion in a Philadelphia-positive CML with marked basophilia: implications for treatment strategy.
Rohon P, Divoka M, Calabkova L, Mojzikova R, Katrincsakova B, Rusinakova Z, Lapcikova A, Raida L, Faber E, Jarosova M, Divoky V, Indrak K.
Rohon P, et al.
Biomed Pap Med Fac Univ Palacky Olomouc Czech Repub. 2011 Jun;155(2):187-90. doi: 10.5507/bp.2011.030.
Biomed Pap Med Fac Univ Palacky Olomouc Czech Repub. 2011.
PMID: 21804629
Free article.
The BCR-ABL mutational status was assessed using sequencing of the RT-PCR products. The in vitro test of sensitivity to TKIs was based on detecting inhibited phosphorylation of the Crkl and Phospho-Src family kinases (SFK, Tyr416) using immunodetection. ...Western blot ana …
The BCR-ABL mutational status was assessed using sequencing of the RT-PCR products. The in vitro test of sensitivity to TKIs was base …
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Two new EPO receptor mutations: truncated EPO receptors are most frequently associated with primary familial and congenital polycythemias.
Kralovics R, Indrak K, Stopka T, Berman BW, Prchal JF, Prchal JT.
Kralovics R, et al.
Blood. 1997 Sep 1;90(5):2057-61.
Blood. 1997.
PMID: 9292543
Free article.
In both cases the EPO dose responses of the erythroid progenitors of the affected subjects were examined to confirm the diagnosis of PFCP. In one of these families, the in vitro behavior of erythroid progenitors in serum-containing cultures without the addition of EPO mimi …
In both cases the EPO dose responses of the erythroid progenitors of the affected subjects were examined to confirm the diagnosis of PFCP. I …
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