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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1965 1
1976 1
1977 1
1979 1
1982 1
1983 1
1984 1
1985 1
1986 1
1990 1
1993 4
1994 2
1995 1
1996 3
1997 4
1998 2
1999 3
2001 2
2002 3
2003 2
2004 4
2005 1
2006 1
2007 5
2008 3
2009 2
2010 4
2011 3
2012 3
2014 8
2015 4
2016 6
2017 6
2018 6
2019 4
2020 7
2021 4
2022 5
2023 4
2024 0

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107 results

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Page 1
Absorbable hemostatic agents.
Gabay M. Gabay M. Am J Health Syst Pharm. 2006 Jul 1;63(13):1244-53. doi: 10.2146/ajhp060003. Am J Health Syst Pharm. 2006. PMID: 16790576 Review.
LEF1-mediated MMP13 gene expression is repressed by SIRT1 in human chondrocytes.
Elayyan J, Lee EJ, Gabay O, Smith CA, Qiq O, Reich E, Mobasheri A, Henrotin Y, Kimber SJ, Dvir-Ginzberg M. Elayyan J, et al. FASEB J. 2017 Jul;31(7):3116-3125. doi: 10.1096/fj.201601253R. Epub 2017 Apr 7. FASEB J. 2017. PMID: 28389425 Free article.
Thus, demonstrating for the first time that SIRT1 represses MMP13 in human OA chondrocytes, which appears to be mediated, at least in part, through repression of the transcription factor LEF1, a known modulator of MMP13 gene expression.-Elayyan, J., Lee, E.-J., Gabay, O., …
Thus, demonstrating for the first time that SIRT1 represses MMP13 in human OA chondrocytes, which appears to be mediated, at least in part, …
Oral Targeted Therapies and Central Nervous System (CNS) Metastases.
Gabay MP, Wirth SM, Stachnik JM, Overley CL, Long KE, Bressler LR, Villano JL. Gabay MP, et al. CNS Drugs. 2015 Nov;29(11):935-52. doi: 10.1007/s40263-015-0283-6. CNS Drugs. 2015. PMID: 26563196 Review.
This includes BRAF inhibitors (dabrafenib and vemurafenib), human epidermal growth factor receptor inhibitors (lapatinib, gefitinib, erlotinib, and afatinib), multi-kinase angiogenesis inhibitors (sorafenib, sunitinib, pazopanib, and vandetanib), and ALK/c-MET (crizotinib) …
This includes BRAF inhibitors (dabrafenib and vemurafenib), human epidermal growth factor receptor inhibitors (lapatinib, gefitinib, erlotin …
Expression and function of junctional adhesion molecule-C in human and experimental arthritis.
Palmer G, Busso N, Aurrand-Lions M, Talabot-Ayer D, Chobaz-Péclat V, Zimmerli C, Hammel P, Imhof BA, Gabay C. Palmer G, et al. Arthritis Res Ther. 2007;9(4):R65. doi: 10.1186/ar2223. Arthritis Res Ther. 2007. PMID: 17612407 Free PMC article.
Junctional adhesion molecule-C (JAM-C) is an adhesion molecule involved in transendothelial migration of leukocytes. ...JAM-C is highly expressed by synovial fibroblasts in RA. Treatment of mice with an anti-JAM-C antibody significantly reduced the sev …
Junctional adhesion molecule-C (JAM-C) is an adhesion molecule involved in transendothelial migration of leukocytes. ...JAM- …
Adaptive immune response in JAM-C-deficient mice: normal initiation but reduced IgG memory.
Zimmerli C, Lee BP, Palmer G, Gabay C, Adams R, Aurrand-Lions M, Imhof BA. Zimmerli C, et al. J Immunol. 2009 Apr 15;182(8):4728-36. doi: 10.4049/jimmunol.0803892. J Immunol. 2009. PMID: 19342649
In the present study, we further examined the adaptive immune responses in JAM-C(-/-) mice. We found that murine conventional dendritic cells express in addition to Mac-1 and CD11c also JAM-B as ligand for JAM-C. By in vitro adhesion assay, we show that murine DCs c …
In the present study, we further examined the adaptive immune responses in JAM-C(-/-) mice. We found that murine conventional dendrit …
Nanotherapy delivery of c-myc inhibitor targets Protumor Macrophages and preserves Antitumor Macrophages in Breast Cancer.
Esser AK, Ross MH, Fontana F, Su X, Gabay A, Fox GC, Xu Y, Xiang J, Schmieder AH, Yang X, Cui G, Scott M, Achilefu S, Chauhan J, Fletcher S, Lanza GM, Weilbaecher KN. Esser AK, et al. Theranostics. 2020 Jun 12;10(17):7510-7526. doi: 10.7150/thno.44523. eCollection 2020. Theranostics. 2020. PMID: 32685002 Free PMC article.
While early therapies sought to deplete all macrophages, current therapeutics aim to reprogram pro-tumor macrophages (M2) and preserve those necessary for anti-tumor immune responses (M1). Recent studies have shown that c-MYC (MYC) is induced in M2 macrophages in vitro and …
While early therapies sought to deplete all macrophages, current therapeutics aim to reprogram pro-tumor macrophages (M2) and preserve those …
Characterization of two azurphil granule proteases with active-site homology to neutrophil elastase.
Wilde CG, Snable JL, Griffith JE, Scott RW. Wilde CG, et al. J Biol Chem. 1990 Feb 5;265(4):2038-41. J Biol Chem. 1990. PMID: 2404977 Free article.
Recently, two additional azurophil granule proteins with NH2-terminal sequence homology to elastase were isolated (Gabay, J. E., Scott, R. W., Campanelli, D., Griffith, J., Wilde, C., Marra, M. N., Seeger, M., and Nathan, C. F. (1989) Proc. Natl. Acad. Sci. U …
Recently, two additional azurophil granule proteins with NH2-terminal sequence homology to elastase were isolated (Gabay, J. E., Scot …
107 results