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Showing results for brain fujikawa
Your search for Brian Fujikawa retrieved no results
[Brain Abscess].
Umeda S, Fujikawa A, Tsuchiya K. Umeda S, et al. No Shinkei Geka. 2021 Mar;49(2):368-374. doi: 10.11477/mf.1436204400. No Shinkei Geka. 2021. PMID: 33762459 Japanese.
Although the prognosis of brain abscesses has historically improved, the mortality rate still ranges from 5 to 32%, with ventricular perforation reaching 50% and 85-100% in fungal brain abscesses. The characteristic finding of ring-like enhancement by contrast-enhan …
Although the prognosis of brain abscesses has historically improved, the mortality rate still ranges from 5 to 32%, with ventricular …
Evidence-based clinical practice guidelines for irritable bowel syndrome 2020.
Fukudo S, Okumura T, Inamori M, Okuyama Y, Kanazawa M, Kamiya T, Sato K, Shiotani A, Naito Y, Fujikawa Y, Hokari R, Masaoka T, Fujimoto K, Kaneko H, Torii A, Matsueda K, Miwa H, Enomoto N, Shimosegawa T, Koike K. Fukudo S, et al. J Gastroenterol. 2021 Mar;56(3):193-217. doi: 10.1007/s00535-020-01746-z. Epub 2021 Feb 4. J Gastroenterol. 2021. PMID: 33538894 Free PMC article.
Higher longitudinal brain white matter atrophy rate in aquaporin-4 IgG-positive NMOSD compared with healthy controls.
Masuda H, Mori M, Hirano S, Uzawa A, Uchida T, Muto M, Ohtani R, Aoki R, Hirano Y; Japanese Alzheimer’s Disease Neuroimaging Initiative (J-ADNI); Kuwabara S. Masuda H, et al. Sci Rep. 2023 Aug 3;13(1):12631. doi: 10.1038/s41598-023-38893-1. Sci Rep. 2023. PMID: 37537208 Free PMC article.
We aimed to compare longitudinal brain atrophy in patients with neuromyelitis optica spectrum disorder (NMOSD) with healthy controls (HCs). ...Patients with AQP4 + NMOSD had a greater annualized NWV atrophy rate than HCs. Suppressing disease activity may prevent brain …
We aimed to compare longitudinal brain atrophy in patients with neuromyelitis optica spectrum disorder (NMOSD) with healthy controls …
Pathophysiological mechanisms of brain damage from status epilepticus.
Wasterlain CG, Fujikawa DG, Penix L, Sankar R. Wasterlain CG, et al. Epilepsia. 1993;34 Suppl 1:S37-53. doi: 10.1111/j.1528-1157.1993.tb05905.x. Epilepsia. 1993. PMID: 8385002 Review.
In very young rats, nonconvulsive normoxic SE spares hippocampal pyramidal cells, but other types of neurons may not show the same resistance, and inhibition of brain growth, DNA and protein synthesis, and of myelin formation and of synaptogenesis may lead to altered br …
In very young rats, nonconvulsive normoxic SE spares hippocampal pyramidal cells, but other types of neurons may not show the same resistanc …
Exercise-induced hypothalamic neuroplasticity: Implications for energy and glucose metabolism.
Hwang E, Portillo B, Grose K, Fujikawa T, Williams KW. Hwang E, et al. Mol Metab. 2023 Jul;73:101745. doi: 10.1016/j.molmet.2023.101745. Epub 2023 May 31. Mol Metab. 2023. PMID: 37268247 Free PMC article. Review.
BACKGROUND: Neuroplasticity refers to the brain's ability to undergo functional and structural changes in response to diverse challenges. Converging evidence supports the notion that exercise serves as a metabolic challenge, triggering the release of multiple factor …
BACKGROUND: Neuroplasticity refers to the brain's ability to undergo functional and structural changes in response to diverse …
Brain glycogen supercompensation following exhaustive exercise.
Matsui T, Ishikawa T, Ito H, Okamoto M, Inoue K, Lee MC, Fujikawa T, Ichitani Y, Kawanaka K, Soya H. Matsui T, et al. J Physiol. 2012 Feb 1;590(3):607-16. doi: 10.1113/jphysiol.2011.217919. Epub 2011 Nov 7. J Physiol. 2012. PMID: 22063629 Free PMC article.
The brain glycogen supercompensation rates after exercise positively correlated with their decrease rates during exercise. ...These results support the hypothesis that, like the effect in skeletal muscles, glycogen supercompensation also occurs in the brain followin …
The brain glycogen supercompensation rates after exercise positively correlated with their decrease rates during exercise. ...These r …
The role of excitotoxic programmed necrosis in acute brain injury.
Fujikawa DG. Fujikawa DG. Comput Struct Biotechnol J. 2015 Mar 28;13:212-21. doi: 10.1016/j.csbj.2015.03.004. eCollection 2015. Comput Struct Biotechnol J. 2015. PMID: 25893083 Free PMC article. Review.
The result of these processes is neuronal necrosis, which is a programmed cell death that is the basis of all acute neuronal injury in the adult brain....
The result of these processes is neuronal necrosis, which is a programmed cell death that is the basis of all acute neuronal injury in the a …
New BBB Model Reveals That IL-6 Blockade Suppressed the BBB Disorder, Preventing Onset of NMOSD.
Takeshita Y, Fujikawa S, Serizawa K, Fujisawa M, Matsuo K, Nemoto J, Shimizu F, Sano Y, Tomizawa-Shinohara H, Miyake S, Ransohoff RM, Kanda T. Takeshita Y, et al. Neurol Neuroimmunol Neuroinflamm. 2021 Oct 19;8(6):e1076. doi: 10.1212/NXI.0000000000001076. Print 2021 Nov. Neurol Neuroimmunol Neuroinflamm. 2021. PMID: 34667128 Free PMC article.
BACKGROUND AND OBJECTIVES: To evaluate the pathophysiology of neuromyelitis optica spectrum disorder (NMOSD) and the therapeutic mechanism and levels of interleukin-6 (IL-6) blockade (satralizumab), especially with respect to blood-brain barrier (BBB) disruption with the n …
BACKGROUND AND OBJECTIVES: To evaluate the pathophysiology of neuromyelitis optica spectrum disorder (NMOSD) and the therapeutic mechanism a …
300 results