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Page 1
Chrom3D: three-dimensional genome modeling from Hi-C and nuclear lamin-genome contacts.
Genome Biol. 2017 Jan 30;18(1):21. doi: 10.1186/s13059-016-1146-2.
Genome Biol. 2017.
PMID: 28137286
Free PMC article.
In Situ Detection of Interactions Between Nuclear Envelope Proteins and Partners.
Barateau A, Buendia B.
Barateau A, et al.
Methods Mol Biol. 2016;1411:147-58. doi: 10.1007/978-1-4939-3530-7_9.
Methods Mol Biol. 2016.
PMID: 27147040
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The p.R482W substitution in A-type lamins deregulates SREBP1 activity in Dunnigan-type familial partial lipodystrophy.
Vadrot N, Duband-Goulet I, Cabet E, Attanda W, Barateau A, Vicart P, Gerbal F, Briand N, Vigouroux C, Oldenburg AR, Lund EG, Collas P, Buendia B.
Vadrot N, et al. Among authors: barateau a.
Hum Mol Genet. 2015 Apr 1;24(7):2096-109. doi: 10.1093/hmg/ddu728. Epub 2014 Dec 18.
Hum Mol Genet. 2015.
PMID: 25524705
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Distinct Fiber Type Signature in Mouse Muscles Expressing a Mutant Lamin A Responsible for Congenital Muscular Dystrophy in a Patient.
Barateau A, Vadrot N, Agbulut O, Vicart P, Batonnet-Pichon S, Buendia B.
Barateau A, et al.
Cells. 2017 Apr 24;6(2):10. doi: 10.3390/cells6020010.
Cells. 2017.
PMID: 28441765
Free PMC article.
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A Novel Lamin A Mutant Responsible for Congenital Muscular Dystrophy Causes Distinct Abnormalities of the Cell Nucleus.
Barateau A, Vadrot N, Vicart P, Ferreiro A, Mayer M, Héron D, Vigouroux C, Buendia B.
Barateau A, et al.
PLoS One. 2017 Jan 26;12(1):e0169189. doi: 10.1371/journal.pone.0169189. eCollection 2017.
PLoS One. 2017.
PMID: 28125586
Free PMC article.
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