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Page 1
NSD2 as a Promising Target in Hematological Disorders.
Int J Mol Sci. 2022 Sep 21;23(19):11075. doi: 10.3390/ijms231911075.
Int J Mol Sci. 2022.
PMID: 36232375
Free PMC article.
Review.
From Loops to Looks: Transcription Factors and Chromatin Organization Shaping Terminal B Cell Differentiation.
Azagra A, Marina-Zárate E, Ramiro AR, Javierre BM, Parra M.
Azagra A, et al.
Trends Immunol. 2020 Jan;41(1):46-60. doi: 10.1016/j.it.2019.11.006. Epub 2019 Dec 7.
Trends Immunol. 2020.
PMID: 31822368
Review.
Item in Clipboard
The HDAC7-TET2 epigenetic axis is essential during early B lymphocyte development.
Azagra A, Meler A, de Barrios O, Tomás-Daza L, Collazo O, Monterde B, Obiols M, Rovirosa L, Vila-Casadesús M, Cabrera-Pasadas M, Gusi-Vives M, Graf T, Varela I, Sardina JL, Javierre BM, Parra M.
Azagra A, et al.
Nucleic Acids Res. 2022 Aug 26;50(15):8471-8490. doi: 10.1093/nar/gkac619.
Nucleic Acids Res. 2022.
PMID: 35904805
Free PMC article.
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HDAC7 is a major contributor in the pathogenesis of infant t(4;11) proB acute lymphoblastic leukemia.
de Barrios O, Galaras A, Trincado JL, Azagra A, Collazo O, Meler A, Agraz-Doblas A, Bueno C, Ballerini P, Cazzaniga G, Stam RW, Varela I, De Lorenzo P, Valsecchi MG, Hatzis P, Menéndez P, Parra M.
de Barrios O, et al. Among authors: azagra a.
Leukemia. 2021 Jul;35(7):2086-2091. doi: 10.1038/s41375-020-01097-x. Epub 2020 Dec 1.
Leukemia. 2021.
PMID: 33262526
No abstract available.
Item in Clipboard
In vivo conditional deletion of HDAC7 reveals its requirement to establish proper B lymphocyte identity and development.
Azagra A, Román-González L, Collazo O, Rodríguez-Ubreva J, de Yébenes VG, Barneda-Zahonero B, Rodríguez J, Castro de Moura M, Grego-Bessa J, Fernández-Duran I, Islam AB, Esteller M, Ramiro AR, Ballestar E, Parra M.
Azagra A, et al.
J Exp Med. 2016 Nov 14;213(12):2591-2601. doi: 10.1084/jem.20150821. Epub 2016 Oct 17.
J Exp Med. 2016.
PMID: 27810920
Free PMC article.
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