Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My NCBI Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1945 1
1946 1
1947 1
1949 1
1950 6
1951 12
1952 15
1953 9
1954 23
1955 11
1956 21
1957 17
1958 10
1959 8
1960 2
1963 8
1964 15
1965 14
1966 14
1967 14
1968 5
1969 10
1970 5
1971 6
1972 3
1973 1
1974 7
1975 7
1976 9
1977 16
1978 28
1979 40
1980 38
1981 41
1982 46
1983 70
1984 67
1985 51
1986 65
1987 62
1988 74
1989 74
1990 65
1991 70
1992 70
1993 58
1994 52
1995 43
1996 46
1997 46
1998 43
1999 41
2000 53
2001 47
2002 30
2003 30
2004 27
2005 36
2006 49
2007 41
2008 46
2009 41
2010 58
2011 68
2012 62
2013 77
2014 65
2015 68
2016 73
2017 68
2018 81
2019 47
2020 110
2021 122
2022 97
2023 99
2024 33

Text availability

Article attribute

Article type

Publication date

Search Results

2,842 results

Results by year

Filters applied: . Clear all
Page 1
The role of macrophages polarization in sepsis-induced acute lung injury.
Wang Z, Wang Z. Wang Z, et al. Front Immunol. 2023 Aug 24;14:1209438. doi: 10.3389/fimmu.2023.1209438. eCollection 2023. Front Immunol. 2023. PMID: 37691951 Free PMC article. Review.
This review provides a comprehensive understanding of the interplay between macrophage polarization and sepsis-induced acute lung injury (SALI) and elaborates on the intervention strategy that centers around the crucial process of macrophage polarization....
This review provides a comprehensive understanding of the interplay between macrophage polarization and sepsis-induced acute lung injury ( …
Be polite.
Ali S. Ali S. CJEM. 2022 Jan;24(1):88-89. doi: 10.1007/s43678-021-00208-6. Epub 2021 Oct 5. CJEM. 2022. PMID: 34611780 No abstract available.
Secretory autophagy during lysosome inhibition (SALI).
Debnath J, Leidal AM. Debnath J, et al. Autophagy. 2022 Oct;18(10):2498-2499. doi: 10.1080/15548627.2022.2095788. Epub 2022 Jul 14. Autophagy. 2022. PMID: 35786367 Free PMC article.
We term this process secretory autophagy during lysosome inhibition (SALI). SALI functionally requires multiple steps in classical autophagosome formation and the small GTPase RAB27A. Because the intracellular accumulation of autophagy cargo receptors perturbs cell …
We term this process secretory autophagy during lysosome inhibition (SALI). SALI functionally requires multiple steps in class …
Circulating mitochondrial DNA-triggered autophagy dysfunction via STING underlies sepsis-related acute lung injury.
Liu Q, Wu J, Zhang X, Li X, Wu X, Zhao Y, Ren J. Liu Q, et al. Cell Death Dis. 2021 Jul 3;12(7):673. doi: 10.1038/s41419-021-03961-9. Cell Death Dis. 2021. PMID: 34218252 Free PMC article.
Whether and how the STING pathway affects autophagic flux during sepsis-induced acute lung injury (sALI) are currently unknown. Here, we demonstrate that the level of circulating mtDNA and degree of STING activation are increased in sALI patients. ...These findings …
Whether and how the STING pathway affects autophagic flux during sepsis-induced acute lung injury (sALI) are currently unknown. Here, …
Koumine regulates macrophage M1/M2 polarization via TSPO, alleviating sepsis-associated liver injury in mice.
Jin GL, Liu HP, Huang YX, Zeng QQ, Chen JX, Lan XB, Xin ZM, Xiong BJ, Yue RC, Yu CX. Jin GL, et al. Phytomedicine. 2022 Dec;107:154484. doi: 10.1016/j.phymed.2022.154484. Epub 2022 Sep 29. Phytomedicine. 2022. PMID: 36215787
However, the protective effect of KM on sepsis-associated liver injury (SALI) and its mechanisms are unknown. PURPOSE: To explore the role of TSPO in SALI and then further explore the protective effect and mechanism of KM on SALI. ...KM, a TSPO ligand, is a p …
However, the protective effect of KM on sepsis-associated liver injury (SALI) and its mechanisms are unknown. PURPOSE: To explore the …
Development of a nomogram for predicting 90-day mortality in patients with sepsis-associated liver injury.
Cui L, Bao J, Yu C, Zhang C, Huang R, Liu L, Shao M. Cui L, et al. Sci Rep. 2023 Mar 4;13(1):3662. doi: 10.1038/s41598-023-30235-5. Sci Rep. 2023. PMID: 36871054 Free PMC article.
The high mortality rate in sepsis patients is related to sepsis-associated liver injury (SALI). We sought to develop an accurate forecasting nomogram to estimate individual 90-day mortality in SALI patients. ...The Kaplan-Meier curves for 90-day survival were differ …
The high mortality rate in sepsis patients is related to sepsis-associated liver injury (SALI). We sought to develop an accurate fore …
Reduning alleviates sepsis-induced acute lung injury by reducing apoptosis of pulmonary microvascular endothelial cells.
Wang Z, Guo Z, Wang X, Liao H, Chen F, Liu Y, Wang Z. Wang Z, et al. Front Immunol. 2023 Jul 3;14:1196350. doi: 10.3389/fimmu.2023.1196350. eCollection 2023. Front Immunol. 2023. PMID: 37465664 Free PMC article.
INTRODUCTION: Sepsis-induced acute lung injury (SALI) is a critical illness with high mortality, and pulmonary microvascular endothelial cells (PMECs) barrier dysfunction is a well-documented pathogenesis of SALI. ...Notably, mTOR has the ability to directly bind to …
INTRODUCTION: Sepsis-induced acute lung injury (SALI) is a critical illness with high mortality, and pulmonary microvascular endothel …
Structural genomics.
Sali A, Lima CD, Kostic M. Sali A, et al. Structure. 2007 Nov;15(11):1341. doi: 10.1016/j.str.2007.10.009. Structure. 2007. PMID: 17997955 Free article. No abstract available.
The SalI (SalGI) restriction-modification system of Streptomyces albus G.
Rodicio MR, Chater KF. Rodicio MR, et al. Gene. 1988 Dec 25;74(1):39-42. doi: 10.1016/0378-1119(88)90246-6. Gene. 1988. PMID: 3074016 Review.
The salIR and salM genes of Streptomyces albus G specify the SalGI (SalI) restriction enzyme and its cognate methyltransferase, respectively. These enzymes are responsible for restriction and modification of bacteriophages. Some phages carry genes that interfere with Sa …
The salIR and salM genes of Streptomyces albus G specify the SalGI (SalI) restriction enzyme and its cognate methyltransferase, respe …
Forsythiaside A Alleviates Lipopolysaccharide-Induced Acute Liver Injury through Inhibiting Endoplasmic Reticulum Stress and NLRP3 Inflammasome Activation.
Fu JN, Liu SC, Chen Y, Zhao J, Lu N, Ma T. Fu JN, et al. Biol Pharm Bull. 2023 Jul 1;46(7):979-986. doi: 10.1248/bpb.b23-00137. Epub 2023 May 13. Biol Pharm Bull. 2023. PMID: 37183023 Free article.
The liver is the primary site of inflammation caused by bacterial endotoxins in sepsis, and septic acute liver injury (SALI) is usually associated with poor outcomes in sepsis. Forsythiaside A (FTA), an active constituent of Forsythia suspensa, has been reported to have an …
The liver is the primary site of inflammation caused by bacterial endotoxins in sepsis, and septic acute liver injury (SALI) is usual …
2,842 results