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Page 1
Suppression of MEK/ERK signaling pathway enhances cisplatin-induced NF-kappaB activation by protein phosphatase 4-mediated NF-kappaB p65 Thr dephosphorylation.
J Biol Chem. 2004 Jun 18;279(25):26143-8. doi: 10.1074/jbc.M402362200. Epub 2004 Apr 8.
J Biol Chem. 2004.
PMID: 15073167
Free article.
Increase of the resistance of human cervical carcinoma cells to cisplatin by inhibition of the MEK to ERK signaling pathway partly via enhancement of anticancer drug-induced NF kappa B activation.
Yeh PY, Chuang SE, Yeh KH, Song YC, Ea CK, Cheng AL.
Yeh PY, et al.
Biochem Pharmacol. 2002 Apr 15;63(8):1423-30. doi: 10.1016/s0006-2952(02)00908-5.
Biochem Pharmacol. 2002.
PMID: 11996883
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Involvement of nuclear transcription factor-kappa B in low-dose doxorubicin-induced drug resistance of cervical carcinoma cells.
Yeh PY, Chuang SE, Yeh KH, Song YC, Cheng AL.
Yeh PY, et al.
Biochem Pharmacol. 2003 Jul 1;66(1):25-33. doi: 10.1016/s0006-2952(03)00250-8.
Biochem Pharmacol. 2003.
PMID: 12818362
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Phosphorylation of p53 on Thr55 by ERK2 is necessary for doxorubicin-induced p53 activation and cell death.
Yeh PY, Chuang SE, Yeh KH, Song YC, Chang LL, Cheng AL.
Yeh PY, et al.
Oncogene. 2004 Apr 29;23(20):3580-8. doi: 10.1038/sj.onc.1207426.
Oncogene. 2004.
PMID: 15116093
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