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Evidence for broad cross-reactivity of the SARS-CoV-2 NSP12-directed CD4+ T-cell response with pre-primed responses directed against common cold coronaviruses.
Westphal T, Mader M, Karsten H, Cords L, Knapp M, Schulte S, Hermanussen L, Peine S, Ditt V, Grifoni A, Addo MM, Huber S, Sette A, Lütgehetmann M, Pischke S, Kwok WW, Sidney J, Schulze Zur Wiesch J. Westphal T, et al. Among authors: schulze zur wiesch j. Front Immunol. 2023 May 5;14:1182504. doi: 10.3389/fimmu.2023.1182504. eCollection 2023. Front Immunol. 2023. PMID: 37215095 Free PMC article.
The magnitude and breadth of hepatitis C virus-specific CD8+ T cells depend on absolute CD4+ T-cell count in individuals coinfected with HIV-1.
Kim AY, Lauer GM, Ouchi K, Addo MM, Lucas M, Schulze Zur Wiesch J, Timm J, Boczanowski M, Duncan JE, Wurcel AG, Casson D, Chung RT, Draenert R, Klenerman P, Walker BD. Kim AY, et al. Among authors: schulze zur wiesch j. Blood. 2005 Feb 1;105(3):1170-8. doi: 10.1182/blood-2004-06-2336. Epub 2004 Sep 30. Blood. 2005. PMID: 15459014 Free article.
Broad repertoire of the CD4+ Th cell response in spontaneously controlled hepatitis C virus infection includes dominant and highly promiscuous epitopes.
Schulze zur Wiesch J, Lauer GM, Day CL, Kim AY, Ouchi K, Duncan JE, Wurcel AG, Timm J, Jones AM, Mothe B, Allen TM, McGovern B, Lewis-Ximenez L, Sidney J, Sette A, Chung RT, Walker BD. Schulze zur Wiesch J, et al. J Immunol. 2005 Sep 15;175(6):3603-13. doi: 10.4049/jimmunol.175.6.3603. J Immunol. 2005. PMID: 16148104
Broadly directed virus-specific CD4+ T cell responses are primed during acute hepatitis C infection, but rapidly disappear from human blood with viral persistence.
Schulze Zur Wiesch J, Ciuffreda D, Lewis-Ximenez L, Kasprowicz V, Nolan BE, Streeck H, Aneja J, Reyor LL, Allen TM, Lohse AW, McGovern B, Chung RT, Kwok WW, Kim AY, Lauer GM. Schulze Zur Wiesch J, et al. J Exp Med. 2012 Jan 16;209(1):61-75. doi: 10.1084/jem.20100388. Epub 2012 Jan 2. J Exp Med. 2012. PMID: 22213804 Free PMC article.
HEV-positive blood donations represent a relevant infection risk for immunosuppressed recipients.
Westhölter D, Hiller J, Denzer U, Polywka S, Ayuk F, Rybczynski M, Horvatits T, Gundlach S, Blöcker J, Schulze Zur Wiesch J, Fischer N, Addo MM, Peine S, Göke B, Lohse AW, Lütgehetmann M, Pischke S. Westhölter D, et al. Among authors: schulze zur wiesch j. J Hepatol. 2018 Jul;69(1):36-42. doi: 10.1016/j.jhep.2018.02.031. Epub 2018 Mar 15. J Hepatol. 2018. PMID: 29551705
Lack of evidence for human serum albumin as major source of HEV infections.
Horvatits T, Westhölter D, Peine S, Schulze Zur Wiesch J, Lohse AW, Lütgehetmann M, Pischke S. Horvatits T, et al. Among authors: schulze zur wiesch j. Transfus Med. 2018 Dec;28(6):470-471. doi: 10.1111/tme.12536. Epub 2018 Apr 30. Transfus Med. 2018. PMID: 29707836 No abstract available.
Detection of a Broad Range of Low-Level Major Histocompatibility Complex Class II-Restricted, Hepatitis Delta Virus (HDV)-Specific T-Cell Responses Regardless of Clinical Status.
Landahl J, Bockmann JH, Scheurich C, Ackermann C, Matzat V, Heide J, Nuurei T, D'Antonio G, von Felden J, Sette A, Peine S, Lohse AW, Luetgehetmann M, Marget M, Sidney J, Schulze Zur Wiesch J. Landahl J, et al. Among authors: schulze zur wiesch j. J Infect Dis. 2019 Jan 29;219(4):568-577. doi: 10.1093/infdis/jiy549. J Infect Dis. 2019. PMID: 30247653
HVEM and CD160: Regulators of Immunopathology During Malaria Blood-Stage.
Muscate F, Stetter N, Schramm C, Schulze Zur Wiesch J, Bosurgi L, Jacobs T. Muscate F, et al. Among authors: schulze zur wiesch j. Front Immunol. 2018 Nov 13;9:2611. doi: 10.3389/fimmu.2018.02611. eCollection 2018. Front Immunol. 2018. PMID: 30483269 Free PMC article.
218 results