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Diverse regional mechanisms drive spinocerebellar ataxia type 1 phenotypes.
Neuron. 2023 Feb 15;111(4):447-449. doi: 10.1016/j.neuron.2023.01.021.
Neuron. 2023.
PMID: 36796325
Free article.
Pathogenetic Mechanisms Underlying Spinocerebellar Ataxia Type 3 Are Altered in Primary Oligodendrocyte Culture.
Schuster KH, Putka AF, McLoughlin HS.
Schuster KH, et al. Among authors: putka af.
Cells. 2022 Aug 22;11(16):2615. doi: 10.3390/cells11162615.
Cells. 2022.
PMID: 36010688
Free PMC article.
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ASOs are an effective treatment for disease-associated oligodendrocyte signatures in premanifest and symptomatic SCA3 mice.
Schuster KH, Zalon AJ, DiFranco DM, Putka AF, Stec NR, Jarrah SI, Naeem A, Haque Z, Zhang H, Guan Y, McLoughlin HS.
Schuster KH, et al. Among authors: putka af.
Mol Ther. 2024 May 1;32(5):1359-1372. doi: 10.1016/j.ymthe.2024.02.033. Epub 2024 Feb 29.
Mol Ther. 2024.
PMID: 38429929
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Disease-associated oligodendrocyte signatures are spatiotemporally dysregulated in spinocerebellar ataxia type 3.
Schuster KH, DiFranco DM, Putka AF, Mato JP, Jarrah SI, Stec NR, Sundararajan VO, McLoughlin HS.
Schuster KH, et al. Among authors: putka af.
Front Neurosci. 2023 Feb 15;17:1118429. doi: 10.3389/fnins.2023.1118429. eCollection 2023.
Front Neurosci. 2023.
PMID: 36875652
Free PMC article.
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Myelinating Glia: Potential Therapeutic Targets in Polyglutamine Spinocerebellar Ataxias.
Putka AF, Mato JP, McLoughlin HS.
Putka AF, et al.
Cells. 2023 Feb 13;12(4):601. doi: 10.3390/cells12040601.
Cells. 2023.
PMID: 36831268
Free PMC article.
Review.
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