Booth DR - Search Results

1

Genetic and transcriptomic analyses support a switch to lytic phase in Epstein Barr virus infection as an important driver in developing Systemic Lupus Erythematosus.

Afrasiabi A, Keane JT, Ong LTC, Alinejad-Rokny H, Fewings NL, Booth DR, Parnell GP, Swaminathan S. Afrasiabi A, et al. Among authors: booth dr. J Autoimmun. 2022 Feb;127:102781. doi: 10.1016/j.jaut.2021.102781. Epub 2021 Dec 21. J Autoimmun. 2022. PMID: 34952359 Free article.

2

Evidence from genome wide association studies implicates reduced control of Epstein-Barr virus infection in multiple sclerosis susceptibility.

Afrasiabi A, Parnell GP, Fewings N, Schibeci SD, Basuki MA, Chandramohan R, Zhou Y, Taylor B, Brown DA, Swaminathan S, McKay FC, Stewart GJ, Booth DR. Afrasiabi A, et al. Among authors: booth dr. Genome Med. 2019 Apr 30;11(1):26. doi: 10.1186/s13073-019-0640-z. Genome Med. 2019. PMID: 31039804 Free PMC article.

3

Transcribed B lymphocyte genes and multiple sclerosis risk genes are underrepresented in Epstein-Barr Virus hypomethylated regions.

Ong LTC, Parnell GP, Afrasiabi A, Stewart GJ, Swaminathan S, Booth DR. Ong LTC, et al. Among authors: booth dr. Genes Immun. 2020 Feb;21(2):91-99. doi: 10.1038/s41435-019-0089-5. Epub 2019 Oct 16. Genes Immun. 2020. PMID: 31619767 Free PMC article.

4

The interaction of Multiple Sclerosis risk loci with Epstein-Barr virus phenotypes implicates the virus in pathogenesis.

Afrasiabi A, Parnell GP, Swaminathan S, Stewart GJ, Booth DR. Afrasiabi A, et al. Among authors: booth dr. Sci Rep. 2020 Jan 13;10(1):193. doi: 10.1038/s41598-019-55850-z. Sci Rep. 2020. PMID: 31932685 Free PMC article.

5

The Interaction of Human and Epstein-Barr Virus miRNAs with Multiple Sclerosis Risk Loci.

Afrasiabi A, Fewings NL, Schibeci SD, Keane JT, Booth DR, Parnell GP, Swaminathan S. Afrasiabi A, et al. Among authors: booth dr. Int J Mol Sci. 2021 Mar 13;22(6):2927. doi: 10.3390/ijms22062927. Int J Mol Sci. 2021. PMID: 33805769 Free PMC article.

6

Gender and the Sex Hormone Estradiol Affect Multiple Sclerosis Risk Gene Expression in Epstein-Barr Virus-Infected B Cells.

Keane JT, Afrasiabi A, Schibeci SD, Fewings N, Parnell GP, Swaminathan S, Booth DR. Keane JT, et al. Among authors: booth dr. Front Immunol. 2021 Sep 8;12:732694. doi: 10.3389/fimmu.2021.732694. eCollection 2021. Front Immunol. 2021. PMID: 34566997 Free PMC article.

7

The interaction of Epstein-Barr virus encoded transcription factor EBNA2 with multiple sclerosis risk loci is dependent on the risk genotype.

Keane JT, Afrasiabi A, Schibeci SD, Swaminathan S, Parnell GP, Booth DR. Keane JT, et al. Among authors: booth dr. EBioMedicine. 2021 Sep;71:103572. doi: 10.1016/j.ebiom.2021.103572. Epub 2021 Sep 3. EBioMedicine. 2021. PMID: 34488019 Free PMC article.

8

The autoimmune risk gene ZMIZ1 is a vitamin D responsive marker of a molecular phenotype of multiple sclerosis.

Fewings NL, Gatt PN, McKay FC, Parnell GP, Schibeci SD, Edwards J, Basuki MA, Goldinger A, Fabis-Pedrini MJ, Kermode AG, Manrique CP, McCauley JL, Nickles D, Baranzini SE, Burke T, Vucic S, Stewart GJ, Booth DR. Fewings NL, et al. Among authors: booth dr. J Autoimmun. 2017 Mar;78:57-69. doi: 10.1016/j.jaut.2016.12.006. Epub 2017 Jan 4. J Autoimmun. 2017. PMID: 28063629

9

Whole blood transcriptomic analysis to identify clinical biomarkers of drug response.

Parnell GP, Booth DR. Parnell GP, et al. Among authors: booth dr. Methods Mol Biol. 2014;1175:35-43. doi: 10.1007/978-1-4939-0956-8_3. Methods Mol Biol. 2014. PMID: 25150865

10

The Multiple Sclerosis (MS) Genetic Risk Factors Indicate both Acquired and Innate Immune Cell Subsets Contribute to MS Pathogenesis and Identify Novel Therapeutic Opportunities.

Parnell GP, Booth DR. Parnell GP, et al. Among authors: booth dr. Front Immunol. 2017 Apr 18;8:425. doi: 10.3389/fimmu.2017.00425. eCollection 2017. Front Immunol. 2017. PMID: 28458668 Free PMC article. Review.

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