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A mechanism linking perinatal 2,3,7,8 tetrachlorodibenzo-p-dioxin exposure to lower urinary tract dysfunction in adulthood.
Turco AE, Oakes SR, Keil Stietz KP, Dunham CL, Joseph DB, Chathurvedula TS, Girardi NM, Schneider AJ, Gawdzik J, Sheftel CM, Wang P, Wang Z, Bjorling DE, Ricke WA, Tang W, Hernandez LL, Keast JR, Bonev AD, Grimes MD, Strand DW, Tykocki NR, Tanguay RL, Peterson RE, Vezina CM. Turco AE, et al. Among authors: tang w. Dis Model Mech. 2021 Jul 1;14(7):dmm049068. doi: 10.1242/dmm.049068. Epub 2021 Jul 27. Dis Model Mech. 2021. PMID: 34318329 Free PMC article.
In utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure exacerbates urinary dysfunction in hormone-treated C57BL/6J mice through a non-malignant mechanism involving proteomic changes in the prostate that differ from those elicited by testosterone and estradiol.
Turco AE, Thomas S, Crawford LK, Tang W, Peterson RE, Li L, Ricke WA, Vezina CM. Turco AE, et al. Among authors: tang w. Am J Clin Exp Urol. 2020 Feb 25;8(1):59-72. eCollection 2020. Am J Clin Exp Urol. 2020. PMID: 32211455 Free PMC article.
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