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High levels of gut-homing immunoglobulin A+ B lymphocytes support the pathogenic role of intestinal mucosal hyperresponsiveness in immunoglobulin A nephropathy patients.
Sallustio F, Curci C, Chaoul N, Fontò G, Lauriero G, Picerno A, Divella C, Di Leo V, De Angelis M, Ben Mkaddem S, Macchia L, Gallone A, Monteiro RC, Pesce F, Gesualdo L. Sallustio F, et al. Among authors: gallone a. Nephrol Dial Transplant. 2021 Feb 20;36(3):452-464. doi: 10.1093/ndt/gfaa264. Nephrol Dial Transplant. 2021. PMID: 33200215 Free PMC article.
A transcriptomics study of hereditary angioedema attacks.
Castellano G, Divella C, Sallustio F, Montinaro V, Curci C, Zanichelli A, Bonanni E, Suffritti C, Caccia S, Bossi F, Gallone A, Schena FP, Gesualdo L, Cicardi M. Castellano G, et al. Among authors: gallone a. J Allergy Clin Immunol. 2018 Sep;142(3):883-891. doi: 10.1016/j.jaci.2018.03.016. Epub 2018 May 4. J Allergy Clin Immunol. 2018. PMID: 29729940 Free article.
Deregulation of autophagy under hyperglycemic conditions is dependent on increased lysine 63 ubiquitination: a candidate mechanism in the progression of diabetic nephropathy.
Pontrelli P, Oranger A, Barozzino M, Divella C, Conserva F, Fiore MG, Rossi R, Papale M, Castellano G, Simone S, Laviola L, Giorgino F, Piscitelli D, Gallone A, Gesualdo L. Pontrelli P, et al. Among authors: gallone a. J Mol Med (Berl). 2018 Jul;96(7):645-659. doi: 10.1007/s00109-018-1656-3. Epub 2018 May 27. J Mol Med (Berl). 2018. PMID: 29806072
Bone marrow fibroblasts overexpress miR-27b and miR-214 in step with multiple myeloma progression, dependent on tumour cell-derived exosomes.
Frassanito MA, Desantis V, Di Marzo L, Craparotta I, Beltrame L, Marchini S, Annese T, Visino F, Arciuli M, Saltarella I, Lamanuzzi A, Solimando AG, Nico B, De Angelis M, Racanelli V, Mariggiò MA, Chiacchio R, Pizzuti M, Gallone A, Fumarulo R, D'Incalci M, Vacca A. Frassanito MA, et al. Among authors: gallone a. J Pathol. 2019 Feb;247(2):241-253. doi: 10.1002/path.5187. J Pathol. 2019. PMID: 30357841
Renal progenitor cells revert LPS-induced endothelial-to-mesenchymal transition by secreting CXCL6, SAA4, and BPIFA2 antiseptic peptides.
Sallustio F, Stasi A, Curci C, Divella C, Picerno A, Franzin R, De Palma G, Rutigliano M, Lucarelli G, Battaglia M, Staffieri F, Crovace A, Pertosa GB, Castellano G, Gallone A, Gesualdo L. Sallustio F, et al. Among authors: gallone a. FASEB J. 2019 Oct;33(10):10753-10766. doi: 10.1096/fj.201900351R. Epub 2019 Jul 3. FASEB J. 2019. PMID: 31268775
Endothelial dysfunction is a hallmark of LPS-induced acute kidney injury (AKI). Endothelial cells (ECs) acquired a fibroblast-like phenotype and contributed to myofibroblast generation through the endothelial-to-mesenchymal transition (EndMT) process. ...B., Castell …
Endothelial dysfunction is a hallmark of LPS-induced acute kidney injury (AKI). Endothelial cells (ECs) acquired a fibroblast- …
A New Vision of IgA Nephropathy: The Missing Link.
Sallustio F, Curci C, Di Leo V, Gallone A, Pesce F, Gesualdo L. Sallustio F, et al. Among authors: gallone a. Int J Mol Sci. 2019 Dec 26;21(1):189. doi: 10.3390/ijms21010189. Int J Mol Sci. 2019. PMID: 31888082 Free PMC article. Review.
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