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Nasopharyngeal carcinoma cells promote regulatory T cell development and suppressive activity via CD70-CD27 interaction.
Gong L, Luo J, Zhang Y, Yang Y, Li S, Fang X, Zhang B, Huang J, Chow LK, Chung D, Huang J, Huang C, Liu Q, Bai L, Tiu YC, Wu P, Wang Y, Tsao GS, Kwong DL, Lee AW, Dai W, Guan XY. Gong L, et al. Among authors: chung d. Nat Commun. 2023 Apr 6;14(1):1912. doi: 10.1038/s41467-023-37614-6. Nat Commun. 2023. PMID: 37024479 Free PMC article.
Peptides designed from molecular modeling studies of the ras-p21 protein induce phenotypic reversion of a pancreatic carcinoma cell line but have no effect on normal pancreatic acinar cell growth.
Kanovsky M, Michl J, Botzolaki G, Morin J, Kovac C, Chung DL, Chie L, Friedman FK, Pincus MR. Kanovsky M, et al. Among authors: chung dl. Cancer Chemother Pharmacol. 2003 Sep;52(3):202-8. doi: 10.1007/s00280-003-0639-3. Epub 2003 May 29. Cancer Chemother Pharmacol. 2003. PMID: 12783204
A protein kinase C inhibitor induces phenotypic reversion of ras-transformed pancreatic cancer cells and cooperatively blocks tumor cell proliferation with an anti- ras peptide.
Way D, Smith S, Sivendran S, Chie L, Kanovsky M, Brandt-Rauf PW, Chung DL, Michl J, Pincus MR. Way D, et al. Among authors: chung dl. Cancer Chemother Pharmacol. 2002 Jun;49(6):429-37. doi: 10.1007/s00280-002-0432-8. Epub 2002 Mar 13. Cancer Chemother Pharmacol. 2002. PMID: 12107546
Induction of oocyte maturation by jun-N-terminal kinase (JNK) on the oncogenic ras-p21 pathway is dependent on the raf-MEK signal transduction pathway.
Chie L, Amar S, Kung HF, Lin MC, Chen H, Chung DL, Adler V, Ronai Z, Friedman FK, Robinson RC, Kovac C, Brandt-Rauf PW, Yamaizumi Z, Michl J, Pincus MR. Chie L, et al. Among authors: chung dl. Cancer Chemother Pharmacol. 2000;45(6):441-9. doi: 10.1007/s002800051017. Cancer Chemother Pharmacol. 2000. PMID: 10854130
16 results